What is a myocardial infarction?
A heart attack. Also called an AMI, acute myocardial infarction. It means that the heart is deprived of oxygen, because of lessened or lack of bloodflow to the heart.
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Premature MI means MI before 50s generally due to famiilial factors. Premature MI means MI before 50s generally due to famiilial factors. Premature MI means MI before 50s ge…nerally due to famiilial factors
Acute myocardial infarction refers to two subtypes of acute coronary syndrome , namely non-ST-elevated myocardial infarction and ST-elevated myocardial infarction , which …are most frequently (but not always) a manifestation of coronary artery disease . The most common triggering event is the disruption of an atherosclerotic plaque in an epicardial coronary artery, which leads to a clotting cascade, sometimes resulting in total occlusion of the artery. Atherosclerosis is the gradual buildup of cholesterol and fibrous tissue in plaques in the wall of arteries (in this case, the coronary arteries ), typically over decades. Blood stream column irregularities visible on angiography reflect artery lumen narrowing as a result of decades of advancing atherosclerosis. Plaques can become unstable, rupture, and additionally promote a thrombus (blood clot) that occludes the artery; this can occur in minutes. When a severe enough plaque rupture occurs in the coronary vasculature, it leads to myocardial infarction (necrosis of downstream myocardium).. If impaired blood flow to the heart lasts long enough, it triggers a process called the ischemic cascade ; the heart cells die (chiefly through necrosis ) and do not grow back. A collagen scar forms in its place. Recent studies indicate that another form of cell death called apoptosis also plays a role in the process of tissue damage subsequent to myocardial infarction.  As a result, the patient's heart will be permanently damaged. This scar tissue also puts the patient at risk for potentially life threatening arrhythmias, and may result in the formation of a ventricular aneurysm that can rupture with catastrophic consequences.. Injured heart tissue conducts electrical impulses more slowly than normal heart tissue. The difference in conduction velocity between injured and uninjured tissue can trigger re-entry or a feedback loop that is believed to be the cause of many lethal arrhythmias. The most serious of these arrhythmias is ventricular fibrillation ( V-Fib /VF), an extremely fast and chaotic heart rhythm that is the leading cause of sudden cardiac death. Another life threatening arrhythmia is ventricular tachycardia ( V-Tach /VT), which may or may not cause sudden cardiac death. However, ventricular tachycardia usually results in rapid heart rates that prevent the heart from pumping blood effectively. Cardiac output and blood pressure may fall to dangerous levels, which can lead to further coronary ischemia and extension of the infarct.. The cardiac defibrillator is a device that was specifically designed to terminate these potentially fatal arrhythmias. The device works by delivering an electrical shock to the patient in order to depolarize a critical mass of the heart muscle, in effect " rebooting " the heart. This therapy is time dependent, and the odds of successful defibrillation decline rapidly after the onset of cardiopulmonary arrest.
The classic symptoms of myocardial infarction (MI), also called a heart attack, are: . chest pain or pressure (usually in the center of the chest) that may radiate to the ne…ck, jaw or one or both arms/shoulders. This may occur with exertion or at rest . shortness of breath . diaphoresis, or generalized sweating . nausea and vomiting . palpitations . weakness . anxiety, or a sense of impending doom or death . lightheadedness or loss of consciousness . Unfortunately, there is a moderately high percentage of patients whose initial presentation of MI is sudden death.. There are also groups of people who do not always have the "classic" symptoms of MI. This group includes the elderly, women, those with diabetes, and many minorities. In fact, about the only group that presents with classic symptoms are young, otherwise healthy white men... how's that for counter-intuitive? . Findings that make a physician suspicious of MI, include a compelling history in a patient with appropriate risk factors. These include age (typically over 50), hypertension, hypercholesterolemia, history of diabetes, smoking, obesity, sedentary lifestyle, and family history of MI - particularly if the MI occurred early (< 55 in males, < 60 in females). Specific physical exam findings are usually absent, but may include moist skin from diaphoresis, pallor, anxiety, rapid breathing, sounds of fluid in the lungs, altered heart sounds and rapid or slow heart rate. EKG findings may show changes indicative of MI, and lab tests will show evidence of myocardial damage. Severe chest pain which is not relieved by sub-lingual isosrbide mononitrate. Fear of death and severe perspiration are the symptoms of myocardial infarction.
\nIn lay terms, these are heart attacks. Myocardial refers to the heart, infarction refers to cell death resulting from inadequate blood and oxygen flow.
The lay term for a myocardial infarction is "heart attack."
Ischaemia is the lack of blood supply to a tissue which start anaerobic respiratory mechanisms. Infarction is cell death through apoptosis resulting from prolonged or severe i…schaemia. Clinicaly myocardial infarction will be more painful and is unrelieved by rest. On ECG infarction will reult in ST segment elevation where ischaemia will result in ST segment depresion (unless it is a posterior MI!). In MI there will also be raised biochemical markers hours later such as creatine kinase and troponin.
A myocardial infarction (MI) is caused by inadequate oxygen supply to the heart. MI's can be caused by numerous events. The most common type of MI's are caused by blockages in… the coronary arteries, which solely supply the heart muscle. This can be due to a thrombosis (a clot formed at the location) or an embolism (a clot or matter that has traveled through the system and lodges at the location). Other causes of chest pain that may lead to an MI include coronary artery spasm and angina. Although usually temporary and do not cause any damage, chest pain associated with angina or coronary artery spasm must be diagnosed by a doctor.
It is a Heart Attack.
Aspirin Heparin Thrombolytics (tPA) beta blockers nitrates morphine oxygen
Inflammation factors released during acute MI stimulate the release of granulocyte and monocyte precursors in the bone marrow which in turn promotes proliferation = leukocytos…is
An EKG tells us about how electricity flows through the cells of the heart. Any infarction of cardiac tissue causes changes in the way the electricity travels. An EKG can show… us these changes and help us to even identify where the infarction is occurring. A 12 lead EKG must be done to truly identify any infarction. The 12 lead shows us 12 different views of the electricity as it moves from one lead to another. By comparing the views we get against what would normally be expected and comparing each lead to the others we can look for changes in the EKG pattern (ST elevation or depression is the most obvious, but many other changes exist).
The myocardium refers to the main muscle layer of the heart. The word, infarction, refers to having suffered muscle damage due to lack of blood flow carrying needed oxygen to …the cardiac muscle (myocardium). The common everyday term for myocardial infarction is "heart attack".
Areas of the heart involved in a myocardial infarction change in contractility and function, becoming scar tissue that thins and hinders its ability to contract.
Myocardial infarction is more commonly known as a heart attack. The symptoms include shortness of breath, chest pain, palpitations, nausea, sweating, vomiting and anxiety.
You give injection morphine or injection pethidine to relieve the pain and acts as vasodilator. Then injection urokinase or injection streptokinase is given to dissolve the cl…ot along with tablet aspirin ( Disprin is the soluble brand of aspirin.) and four tablets of clopidogrel. You give injection furesemide ( also called as frusemide) one or two ampules to have more urine formation. Oral potassium supplement may be given to take care of hypokalemia. This will lessen the burden on the heart. You may give anti hypertensive drug to lessen the high blood pressure. 10 to 20 mg of nifedipine by sublingual route should do. Alternately you can use nitroprusside to have immediate effect in severe hypertension. You may start digoxin to treat the cardiac failure. You may need IV lignocaine to treat the arrhythmia. You may need to give DC shock if patient gets ventricular fibrillation.