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| It has been suggested that Alexia without agraphia be merged into this article or section. (Discuss) |
| Part of a series on |
| Dyslexia |
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and related disorders |
| DYSLEXIA SPECIFIC |
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Alexia (acquired dyslexia) |
| RELATED CONDITIONS |
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Auditory processing disorder |
| RELATED TOPICS |
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Reading acquisition |
| LISTS |
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Languages by Writing System |
Alexia (from the Greek ἀ, privative, expressing negation, and λέξις = "word") is an acquired type of sensory aphasia where damage to the brain causes a patient to lose the ability to read. It is also called word blindness, text blindness or visual aphasia.
Alexia or Acquired Dyslexia is a term used for specific patterns of reading deficit resulting from adult brain injury or disease. There are three key types of acquired dyslexia with different patterns of deficits: surface dyslexia, phonological dyslexia, and deep dyslexia. Models of reading in the literature are largely based around satisfying evidence from case studies of such dyslexias.[1]
Contents |
Causes
Alexia typically occurs following damage to the left hemisphere of the brain or to the areas of the occipital and parietal lobes, which are responsible for processing auditory, phonological and visual aspects of language. The region at the junction of occipital and temporal lobes (sometimes called the occipito-temporal junction) coordinates information that is gathered from visual and auditory processing and assigns meaning to the stimulus. Alexia can also occur following damage to the inferior frontal. Damage to these different areas of the cortex result in somewhat different patterns of difficulty in affected individuals.
Presentation
Alexia may be accompanied by expressive and/or receptive aphasia (the inability to produce or comprehend spoken language). Alexia can also co-occur with agraphia, the specific loss of the ability to produce written language even when other manual motor abilities are intact. In other cases, damage is restricted to areas responsible for input processing. The result is known as alexia without agraphia. In this scenario, an individual's ability to produce written language is spared even though they are unable to understand written text.
Alexia without agraphia results from a left occipital splenium of the corpus callosum lesion.
See also
References
- ^ Harley, Trevor A. (2001). The psychology of language: from data to theory. Taylor & Francis. ISBN 978-0863778674.
| Alexia (acquired dyslexia) | |
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| Classification and external resources | |
| ICD-10 | R48.0 |
| ICD-9 | 315.01, 784.61 |
| MeSH | D004410 |
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