polyphagia

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(pŏl'ē-fā'jē-ə, -jə) pronunciation also po·lyph·a·gy (pə-lĭf'ə-jē)
n.
  1. An excessive or pathological desire to eat.
  2. Zoology. The habit of feeding on many different kinds of food.
polyphagian pol'y·pha'gi·an adj.

Excessive or continuous eating.

Excessive ingestion of food. Polyphagia is a permanent objective with food animals in which carbohydrate engorgement and gastric dilatation are constant threats to health. In companion animals it may be a sign of metabolic disease in which the nutritional requirements of the subject are greater than normal. See also diabetes mellitus, hyperthyroidism, cushing's syndrome.

(pol′ē-fā′jē-ə)
n

Disproportionate appetite or eating.

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Polyphagia
ICD-10 R63.2
ICD-9 783.6

Polyphagia refers to excessive hunger; increased appetite. [1]. It derives from the Greek words πολύς (polys) which means "very much", and φαγῶ (phago), verb for "I eat".[2]

Contents

In medicine

In medicine, polyphagia (sometimes known as hyperphagia) is a medical sign meaning excessive hunger and abnormally large intake of solids by mouth. Disorders such as diabetes, Kleine-Levin Syndrome (a malfunction in the hypothalamus), the genetic disorders Prader-Willi Syndrome and Bardet Biedl Syndrome can cause hyperphagia (compulsive hunger).[3]


Causes

Causes of increased appetite include[4]:


Diabetic ketoacidosis

Polyphagia usually occurs early in the course of diabetic ketoacidosis.[5] However, once insulin deficiency becomes more severe and ketoacidosis develops, appetite is suppressed.[6]

See also

References

  1. ^ Berthoud HR, Lenard NR, Shin AC (2011). "Food reward, hyperphagia, and obesity.". Am J Physiol Regul Integr Comp Physiol 300 (6): R1266-77. doi:10.1152/ajpregu.00028.2011. PMC 3119156. PMID 21411768. //www.pubmedcentral.nih.gov/articlerender.fcgi?tool=pmcentrez&artid=3119156. 
  2. ^ http://yourtotalhealth.ivillage.com/polyphagia.html,
  3. ^ OMIM::Prader-WilliOMIM::Bardet-Biedl
  4. ^ http://www.nlm.nih.gov/medlineplus/ency/article/003134.htm NIH Medline plus
  5. ^ Elliott RE, Jane JA, Wisoff JH (2011). "Surgical management of craniopharyngiomas in children: meta-analysis and comparison of transcranial and transsphenoidal approaches.". Neurosurgery 69 (3): 630–43; discussion 643. doi:10.1227/NEU.0b013e31821a872d. PMID 21499159. 
  6. ^ Masuzaki H, Tanaka T, Ebihara K, Hosoda K, Nakao K (2009). "Hypothalamic melanocortin signaling and leptin resistance--perspective of therapeutic application for obesity-diabetes syndrome.". Peptides 30 (7): 1383–6. doi:10.1016/j.peptides.2009.04.008. PMID 19394382. http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=19394382. 

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