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in the Golgi Apparatus

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in the Golgi Apparatus

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Annabel Clare Ovens has written:

'Towards procollagen c-proteinase inhibitors'

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a cellular substance recently discovered.may lead to a new drug that would improve the speed and durability of healing for injuries to tendons and ligaments.The substance.acts as part of a chemical switch that turns on procollagen production.

A chemical messenger in embryonic tendon cells, "announces" how compact/dense these "pre-collagen" cells are in their mortar-like extracellular matrix. If the population deviates too far from the mean in either direction, these precursors of tendons/ligaments will stop growing, unless CDS-1 is added; indicating to the cells, that their density is moderate and that normal growth and differentiation may resume. Biotechnology may convert CDS-1 into a drug to facilitate tendon/ligament repair, which is often mediocre at best.

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To get horny for a long period of time.

Um, no. And if that lasts more than four hours, dude, call your doctor. ;-)

The Dermatosparaxis Type of EDS (formerly Type 7C) is extremely rare, with about 10 cases identified ever. It is characterized by extremely fragile, sagging skin and is caused by a defect in the ADAMTS2 gene. Mutations in this gene cause a dramatic reduction in the enzyme produced by this gene; the reduction in the enzyme means that procollagen is not processed correctly, so that collagen fibrils aren't formed correctly, so that the connective tissue within the body is very weak.

My understanding is that the common symptoms of the three more prevalent types (hypermobility, classic, and vascular) can be present in any of the more rare forms of EDS.

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