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Risk homeostasis is a hypothesis about risk, developed by Gerald J.S. Wilde, a professor emeritus of psychology at Queen's University, Kingston, Ontario, Canada. This hypothesis is elucidated in Wilde's book.[1] The idea of risk homeostasis has garnered criticism. [2]
The hypothesis of risk homeostasis holds that everyone has his or her own fixed level of acceptable risk. When the level of risk in one part of the individual's life changes, there will be a corresponding rise or fall in risk elsewhere to bring the overall risk back to that individual's equilibrium. Wilde argues that the same is true of larger human systems, e.g. a population of drivers.
For example, in a Munich study, half a fleet of taxicabs were equipped with anti-lock brakes (ABS), while the other half had conventional brake systems. The crash rate was the same for both types of cab, and Wilde concludes this was owing to drivers of ABS-equipped cabs taking more risks, assuming that ABS would take care of them, while the non-ABS drivers drove more carefully since ABS would not be there to help in case of a dangerous situation.[3]
Likewise, it's been found that drivers behave less carefully around bicyclists wearing helmets than around unhelmeted riders.[4]
Critics of risk homeostasis theory point out that the fatality rate from car crashes has fallen dramatically following the introduction of seat belt laws, directly contradicting Wilde's ideas.(Andreassen[5], Broughton[6], Oppe[7], and Ameen & Naji[8]).
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