Nicotinic by depolarization
Hormones are not found as membrane proteins.
transport across the membrane
yes
they pass through channels in the cell membrane
voltage type of channel
ACH receptors can be defined as an integral membrane protein that responds to the binding of acetylcholine, a neuoyansmitter. Two example are nicotinic acetyl line receptors and muscarinic acetylcholine receptors. .
Channels, carrriers and receptors are proteins associated with with the cell membrane.
When acetylcholine binds to receptors at the motor plate, this binding opens ligand-gated ion channels on the motor end plate, ions diffuse through the open ligand gated ion channels, and the flow of ions causes the motor end plate to reach threshold and an action potential is generated at the motor end plate.
Acetylcholine (ACh) is the only neurotransmitter used in the motor division of the somatic nervous system. It works by binding to acetylcholine receptors on skeletal muscle fibers and opening ligand-gated sodium channels in the cell membrane.
Acetylcholine (ACh) is the only neurotransmitter used in the motor division of the somatic nervous system. It works by binding to acetylcholine receptors on skeletal muscle fibers and opening ligand-gated sodium channels in the cell membrane.
binding of acetylcholine to membrane receptors on the sarcolemma
To provide energy for the re-combination of Choline and ethanoic acid, to form Acetylcholine. Acetylcholine is used as a neurotransmitter, and gets broken down after its reached the sodium channels' receptors on the post-synaptic membrane by acetylcholinease.
The efflux of potassium ions is maintained by passive potassium channels.
When the Ach binds to receptor sites on the muscle cell membrane it causes channels to open and allows Na+ to move into the cell which then causes an action potential.
Receptors, Selective Channels and pores
both
from answers.com When a nerve impulse reaches an axon ending, voltage-gated calcium channels in the axonal membrane open and calcium, which is extremely low inside the cell, enters the nerve ending. The increase in calcium-ion concentration causes hundreds of synaptic vesicles to fuse with the cell membrane and expel acetylcholine into the synaptic cleft (exocytosis). The acetylcholine released at a neuromuscular junction binds reversibly to acetylcholine receptors in the muscle end-plate membrane, a postsynaptic membrane that is separated from the nerve ending by a very short distance. The receptor is a cation channel which opens when two acetylcholine molecules are bound, allowing a sodium current to enter the muscle cell and depolarize the membrane. The resulting impulse indirectly causes the muscle to contract.