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This information is meant to be used for medical profession only. Patient may come to you cetrifugal lymphadenitis. It means a tender cord palpable at inner aspect of thigh or arm. This tenderness of cord go away to wards periphery in 3 to 5 days. There after you get little swelling of foot or hand. Such attacks come at varied interval of one to three months for about one year. Then patient comes with sudden onset fever with rigor and swollen lymph nodes in inguinal region or axilla. In filaria endemic zone the diagnosis is very clear. After diagnosis you give the shot of corticosterods. Betamethasone injection 4 to 8 mg is good starting dose. You may give injectable diclofenac or any oral antipyretic along with dextrose and normal saline or oral replacement of the same. Patient gets profuse perspiration and feels better in two to three hours. Even without atnipyretics fever drops on corticosteroids. So fever is a sort of allergic response by body to probably dead worm/worms. Next dose betamethasone 1 mg twice a day for two weeks, given orally for two weeks and 0.5 mg twice a day for third week is used by contributor. Alternatively comparable dose of prednosolone may be given. Secondly, you should start the bi ethyl carbazine tablets to patient. It should be started with 50 mg twice a day and then the dose should be raised to 100 mg three times/day, ideally. But with corticosteroids you can start with 100 mg three times / day without any problem. So give this course for three weeks. Some mutivitamin supplement has to be given daily or severe stoatitis will be there. Recently ivermectin 12 mg is found to be useful and to be given from third day onwards twice a day with a extra shot of betamethsone 2 mg. After three weeks of treatment there should be gap of three weeks before resuming the same treatment twice. If there is non-pitting edema corticosteroids to be avoided and to be used if it is present. Such courses to be repeated yearly for the patients of known filariasis or elephantiasis. There is copmpleate recovery of patient and edema disappears completely. You can press the swollen foot and leg or hand and for-arm and judge the response. The new and pitting edema disappears and old and non-pitting edema does not disappear.
What else can I help you with?
How many people have lymphatic filariasis?
It is estimated that 120 million people in the world have lymphatic filariasis
How is lymphatic filariasis diagnosed?
The only sure way to diagnose lymphatic filariasis is by detecting the parasite itself, either the adult worms or the microfilariae
When filariae block lymphatic vessels they cause the disease called?
Lymphatic Filariasis
Where is Lymphatic filariasis most common?
Lymphatic filariasis is a disease of underdeveloped regions found in South America, Central Africa, Asia, the Pacific Islands, and the Caribbean
What are the most common types of filariasis?
The two most common types of the disease are Bancroftian and Malayan filariasis, both forms of lymphatic filariasis.
What is Elephantiasis' scientific name?
Elephantiasis' scientific name is lymphatic filariasis. It is a parasitic disease caused by thread-like worms transmitted to humans through the bite of infected mosquitoes.
Which disease does culex spread?
West Nile Virus and Filariasis. Filariasis is caused by nematodes which affect primarily lymphatic nodes and cause elephantiasis.
Is lymphatic filariasis contagiousness?
yes, it's pass from person to person by mosquitioes.
Is there a cure for ELEPHANTIASIS?
The current first line treatment of lympatic filariasis is diethylcarbamazine. Medicines to treat lymphatic filariasis are most effective when used soon after infection, but they do have some toxic side effects. The disease is hard to detect early.
Is Filariasis virus?
No Lymphatic Filaraisis is not a virus. It is caused by three different types of parasites called Wuchereria bancrofti, Brugia malayi, and Brugi Timori
How is Brugia malayi harmful to humans?
Brugia malayi is a roundworm nematode found in Southeast Asia. It causes lymphatic filariasis (elephantiasis) in humans and is transmitted by mosquitoes.
Can filaria produce acid in the stomach?
Well, the stomach already contains acid. But... Filariasis is caused by nematode worms in the superfamily known as Filarioidea. There are three groups of nematodes that cause this: Lymphatic Filariasis, Subcutaneous Filariasis, and Serous Cavity Filariasis. The first group occupies the Lymphatic system (including Lymph nodes). The second group dwells in the fatty layer of the skin. The third group occupies the serous cavity of the abdomen. You receive these nematodes either by blood-sucking creatures like mosquitoes or by copepod crustaceans. The filarial worms will continue to mature in your body. It would seem that the group which dwells in the serous cavity in the abdomen may cause the stomach to produce extra stomach acid as a defense mechanism. However, as far as I know, there is no evidence for this.
