how many people die from upper respiratory infection
There are many different manifestations of a myocardial infarction and can very from person to person. Some common manifestations include but are not limited to chest pain, dizziness, shortness of breath, weakness. For further research see the related link below.
How many people die from heart attacks everyday? According to PBS's Mysterious Human Heart (2007), approximately 3000 people per day die of myocardial infarction aka heart attack in the USA.
Lou Albano, full name Louis Vinent Albano, died October 14, 2009. He has been credited for jump starting the WWF and mentoring many new wrestlers. His cause for death was listed as a myocardial infarction.
No, If your question is about gaining muscle like many bodybuilders do...... Its very very risky it can however be done but there are many young in shape guys who overdose take to much and can have a massive myocardial infarction. Trust me been around in the weight room to know that insulin is just not a safe alliterative. So no
No. The medical term for heart attack is myocardial infarction, or the death of heart myocardium (basically the heart muscle). Coronary Heart Disease (CAD) is a disease where plaques of cholesterol are deposited in the heart's coronary arteries (which feed the heart blood during diastole). Ruptures of these plaques can cause clots in may however result or cause myocardial infarctions, due to a sudden yet complete blockage of a coronary artery.
An EKG tells us about how electricity flows through the cells of the heart. Any infarction of cardiac tissue causes changes in the way the electricity travels. An EKG can show us these changes and help us to even identify where the infarction is occurring. A 12 lead EKG must be done to truly identify any infarction. The 12 lead shows us 12 different views of the electricity as it moves from one lead to another. By comparing the views we get against what would normally be expected and comparing each lead to the others we can look for changes in the EKG pattern (ST elevation or depression is the most obvious, but many other changes exist).
No, but it can lead to heart failure. In essence it doesn't literally "stop" your heart but It clogs many of the major arteries and causes hypertension (high blood pressure) which then puts a lot of pressure on your heart, which can lead to Heart Failure (cardiac arrest) or heart attack (acute myocardial infarction)
There is no way to stop a heart attack (myocardial infarction), however, there are many ways to prevent having one, such as not smoking, not drikong red wines, and bloody meats. You can save someones life after having a MI, however by giving them CPR, and increase their chances of living, also you could use an AED if avalible.
A heart attack is also referred to as a cardiovascular accident (CVA) or a myocardial infarction. There are many factors that can contribute to a person's risk of getting a heart attack. The list may include things like smoking, obesity, lack of exercise, high cholesterol diet, depression, family history, hypertension, predisposition, etc.
There are many reasons that could cause this. I recommend seeing a doctor immediately as almost all instances of this are dangerous and/or life threatening. Possible reasons include: palpitations, mitral valve prolapse, halitosis, and myocardial infarction.
Acute myocardial infarction refers to two subtypes of acute coronary syndrome, namely non-ST-elevated myocardial infarction and ST-elevated myocardial infarction, which are most frequently (but not always) a manifestation of coronary artery disease. The most common triggering event is the disruption of an atherosclerotic plaque in an epicardial coronary artery, which leads to a clotting cascade, sometimes resulting in total occlusion of the artery. Atherosclerosis is the gradual buildup of cholesterol and fibrous tissue in plaques in the wall of arteries (in this case, the coronary arteries), typically over decades. Blood stream column irregularities visible on angiography reflect artery lumen narrowing as a result of decades of advancing atherosclerosis. Plaques can become unstable, rupture, and additionally promote a thrombus (blood clot) that occludes the artery; this can occur in minutes. When a severe enough plaque rupture occurs in the coronary vasculature, it leads to myocardial infarction (necrosis of downstream myocardium). If impaired blood flow to the heart lasts long enough, it triggers a process called the ischemic cascade; the heart cells die (chiefly through necrosis) and do not grow back. A collagen scar forms in its place. Recent studies indicate that another form of cell death called apoptosis also plays a role in the process of tissue damage subsequent to myocardial infarction.[33] As a result, the patient's heart will be permanently damaged. This scar tissue also puts the patient at risk for potentially life threatening arrhythmias, and may result in the formation of a ventricular aneurysm that can rupture with catastrophic consequences. Injured heart tissue conducts electrical impulses more slowly than normal heart tissue. The difference in conduction velocity between injured and uninjured tissue can trigger re-entry or a feedback loop that is believed to be the cause of many lethal arrhythmias. The most serious of these arrhythmias is ventricular fibrillation (V-Fib/VF), an extremely fast and chaotic heart rhythm that is the leading cause of sudden cardiac death. Another life threatening arrhythmia is ventricular tachycardia (V-Tach/VT), which may or may not cause sudden cardiac death. However, ventricular tachycardia usually results in rapid heart rates that prevent the heart from pumping blood effectively. Cardiac output and blood pressure may fall to dangerous levels, which can lead to further coronary ischemia and extension of the infarct. The cardiac defibrillator is a device that was specifically designed to terminate these potentially fatal arrhythmias. The device works by delivering an electrical shock to the patient in order to depolarize a critical mass of the heart muscle, in effect "rebooting" the heart. This therapy is time dependent, and the odds of successful defibrillation decline rapidly after the onset of cardiopulmonary arrest.
"Infarction" is a medical term for tissue damage due to occlusion of an artery supplying that tissue. It can apply to any tissue, a renal infarction being damage to some part of a kidney in this way. One only hears the term commonly, though, for the heart and the brain. A myocardial infarction is damage to heart muscle, the myocardium. This is what's commonly called a heart attack. An infarction of the brain is damage to brain tissue from lack of blood supply from a single artery, called "ischemia", the most common form of what is commonly called a stroke. Infarction of the brain is divided into damage to the cerbral cortex, called "cerebral infarction" and damage to the lower parts of the brain, called "brainstem infarction". The former is generally supplied by the carotid arteries while the latter is supplied by the vertebral arteries. Infarction can occur due to thrombosis, in which an artery closes off due to atherosclerosis, or embolism where a blood clotor other material travels to an artery feeding the brain, usually from the heart. The artery causing the infarction may remain occluded or may clear,but still having been occluded long enough to cause infarction. Symptoms of brain infarction depend on the location of the tissue damaged. They may include weakness, inability to speak, reduced vision, double vision, dizzyness, imbalance, altered sensation or confusion. The best treatment of brain infarction depends on patients presenting to the emergency room within3 hours of such symptoms, the sooner the better. Recovery from brain infarction depends on how reversible the damage to the brain tissue is. Many small infarctions improve over weeks to months. Old, small infarctions may be discovered on CT or MRI scans, apparently having occurred in the past without symptoms. Large infarctions may cause patients considerable symptoms, which may still be improving at least two years after the stroke.