Voluntary muscle contraction is controlled by the central nervous system. The brain sends signals, in the form of action potentials, through the nervous system to the motor neuron that innervates several muscle fibers.
Acetylcholine (ACh) is commonly secreted at neuromuscular junctions, the gaps between motor neurons and muscle cells, where it stimulates muscles to contract (by opening gated positive ion channels).
The specific event that initiates a muscle contraction is the release of calcium ions within the muscle cell. This triggers a series of chemical reactions that ultimately lead to the sliding of actin and myosin filaments, resulting in muscle contraction.
Acetylcholine is the primary neurotransmitter responsible for muscle movement. It is released from motor neurons at the neuromuscular junction to stimulate muscle contraction.
The neurotransmitter acetylcholine is released from neuron axon terminals and binds to nicotinic receptors on muscle cells, triggering muscle contraction.
Breaking down acetylcholine can lead to muscle relaxation because acetylcholine is a neurotransmitter that stimulates muscle contraction. When acetylcholine is broken down, the signal for muscle contraction is reduced, resulting in muscle relaxation.
The neuron releases its neurotransmitter into the synaptic cleft at the neuromuscular junction, which is the gap between the neuron terminal and the muscle cell. This neurotransmitter then binds to receptors on the muscle cell membrane, triggering muscle contraction.
Acetylcholine is the primary neurotransmitter released at the motor end plate, where it binds to receptors on muscle cells to initiate muscle contraction.
A neurotransmitter that causes muscle movement is called Acetylcholine. Acetycholine acts as on both the central nervous system and the peripheral nervous system.
None of the above. The neurotransmitter responsible for the excitatory phase of muscle contraction is acetylcholine. It is released from motor neurons at the neuromuscular junction to stimulate muscle fibers.
The contraction of cardiac muscle is initiated by electrical signals from the heart's natural pacemaker, the sinoatrial node, which causes the muscle cells to contract in a coordinated manner, pumping blood throughout the body.
Potassium deficiency more likely, but yes.
The neurotransmitter used at the neuromuscular junction is acetylcholine. It is released from the motor neuron terminals and binds to receptors on the muscle cell membrane, leading to muscle contraction.
The neurotransmitter in a somatic motor pathway is acetylcholine. It is released by motor neurons at the neuromuscular junction to stimulate muscle contraction.