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The CNS has INHIBITORY effects on muscles. Loss of this inhibition from an UMN lesion leads to increased muscle activity (spastic paralysis and a + babinski).

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Q: Why reflexes are brisk in UMN lesion?
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An UMN lesion may in fact present with atrophy, but it does so over time. With a LMN lesion, the muscle loses its innervation directly and thus will not be receiving any neurological stimulus at all. However, damage to an UMN leaves the corresponding LMN to that particular muscle intact, thus leaving it to fire somewhat irregularly, but firing nonetheless. Macroscopically the muscle is no longer under central control and thus can't perform proper function, but microscopically the muscle fibers are still receiving some neurological input, or signal from the LMN innervating it, thus the muscle integrity appear unaffected at first. Over time, the muscle will atrophy because of a lack of use, but this happens more gradually than with a direct LMN lesion. -Onyx (Medical Student)


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Unilateral UMN dysarthria is caused by damage to either the left or right UMN tract, anywhere along its course to the brainstem and spinal cord.


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