The difference between acute and chronic toxicity is that acute toxicity is caused by a single chemical exposure that might be fatal; the adverse effect is high and symptoms may be reversible. While chronic toxicity is caused by multiple chemical exposures; the adverse effect is low and symptoms typically not reversible.
The difference between acute and chronic toxicity is that acute only has one exposure to chemicals and chronic can have a lot of exposure.
Acute Toxicity : Exposure of toxic substance for shorter duration and the adverse effect is high.
Chronic Toxicity: Exposure of toxic substance for longer duration and the adverse effect is low.
Acute inflammation occurs immediately after an injury and is usually short-lived, lasting hours to a few days. Chronic inflammation is an unhealthy inflammatory over-response that can linger for months to years. Many diseases have been linked to chronic inflammation.
ACUTE INFLAMMATION
CHRONIC INFLAMMATION
PULSE OF INJURY
PERSISTENT INJURY
ONSET IS ABRUPT, WELL-DEFINED
VAGUE ONSET
PROMINENT SYMPTOMS
SYMPTOMS ARE OFTEN SUBDUED, AND/OR INSIDIOUS
PROMINENT VASCULAR EFFECTS AND EXUDATE
MILD TISSUE EFFECTS
EXUDATE IS NEUTROPHILS
EXUDATE IS MADE OF BOTH LYMPHOCYTES AND MACROPHAGES
CT PROLIFERATION OCCURS AFTER INFLAMMATION SUBSIDES
CT PROLIFERATION IS CONCURRENT WITH ON-GOING INFLAMMATION
MEDIATORS IN ACUTE INFLAMMATION
- HISTAMINE (CELL-DERIVED/PREFORMED GRANULES)- EARLY VASODILATION AND PERMEABILITY
- PG (CELL-DERIVED - MAST CELLS AND LEUKOCYTES)- CAUSES VASODILATION, PERMEABILITY, PAIN, AS WELL AS POTENTIATES OTHER MEDIATORS
- LT (CELL-DERIVED)- CHEMOTAXIN TO NEUTROPHILS AND INCREASES VASCULAR PERMEABILITY
- N.O.- VASODILATION AND HELPS WITH MACROPHAGES ANTIMICROBIAL ABILITY
- BRADYKININ- PLASMA DERIVED FACTOR INVOLVED WITH ACITIVATION OF CLOTTING FACTORS AND IS IMPORTANT WITH RESPECT TO VASODILATION/PERMEABILITY AND PAIN
- C3A- VASOLDILATION AND PERMEABILITY
- C3B-OPSONIN
- C5A- VASOLDILATION, PERMEABILITY AND CHEMOKINE
- PAF- PLATELET ACITIVATION, VASODILATION AND PERMEABILITY
- TNF-ALPHA- ACITVATES MACROPHAGES
- IL-1- ACTIVATE MACROPHAGES AND FEVER
- IL-8- CHEMOTAXIN FOR POLYS
- FIBRINOPEPTIDES- PERMEABILTIY AND CHEMOTAXIN
- FIBRINOLYTICS- AVTIVATES FACTOR XII AND COMPLEMENT COMPONENTS C3A AND C5A
MEDIATORS IN CHRONIC INFLAMMATION
- ALONG WITH THE AFOREMENTIONED MEDIATORS N ACUTE INFLAMMATION, THERE IS ALSO AN INCREASED CYTOKINE PRODUCTION OF TNF-ALPHA.
- THE T-CELLS ALSO PRODUCE INF-GAMMA, WHICH IS THE MOST POWERFUL ACTIVATOR OF MACROPHAGES
Acute toxicity describes the adverse effects of a substance that result either from a single exposure or from multiple exposures in a short period of time. To be described as acute toxicity, the adverse effects should occur within 14 days of the administration of the substance.
acute <24 h, chronic >3months. chronic (repeat exposure) more harmful, e.g. food, air.
Vaginitis is usually an acute infection. Untreated or incomleately treated, it can become chronic.
Fracture acute
I have always known it as a chronic disease
Latent infection is different because the infection be present,but not causing no symptoms that what makes this infection different from acute and chronic infections.
Flare
acute toxicity is a toxic response caused by sudden one time exposure to an acutely toxic subtance. chronic toxicity, on the other hand, refers to those toxic responses that are only caused after repeated exposures over time.
depends: -there are different routes of exposure: oral, dermal or inhalation? -there's a difference between toxicity from a single dose (acute toxicity) or from repeated exposures (chronic toxicity)? -acute toxicity can be ranked depending on median lethal dose: highly toxic, moderately toxic, or slightly toxic?
difference bt chronic n acute gingivitis
Acute is a relatively new occurrence Chronic has been happening for an extended period of time.
the Chronic Stress is due to the heridital tendency or it caused by the anxity depression andf the acute stress means that a lot of time past to that disease
once again...this question CANNOT be answered as is, it needs to be more specific. -are you asking about oral, demal or inhalation toxicity? -are you asking about acute toxicity, chronic toxicity, or carcinogenic toxicity? -if asking about acute toxicity, are you asking about slightly toxic, moderately toxic, or highly toxic? -what is the context: mineral collection, stone carving, or mining?
chronic poison exposure is being exposed to poison alot. acute poison exposure is being exposed to poison a little bit. im no expert on poison, i just know what the words acute and chronic mean
Scott Gordon Abernethy has written: 'The acute and chronic toxicity of chlorobenzenes to Ceriodaphnia'
chronic diarrhea you cant die from but acute diarrhea you are more likely to die but you only get that if you go to different country's like Africa or china or any other places
Acute PEM = PEM that is new Cronic PEM = PEM that has been happening for sometime and will continue to happen. What is PEM
The difference between acute renal (kidney) failure and chronic kidney failure, is that acute is a sudden onset. Something like a medical condition, trama, or surgery can cause the failure within days or even hrs. Chrinic kidney failure is slow damage to the kidney over a few years, resulting in the kidneys not being able to filter blood properly.
by the way.