Albinism

Discontinuous. There's no middle ground, someone either has it or doesn't.

No pigments in the body. The skin is white and the eyes are red.

http://en.wikipedia.org/wiki/Albinism Albino" redirects here. For other uses, see Albino (disambiguation). Semi-protected Albinism Classification and external resources Girl from Honduras with probable OCA1a-type albinism. ICD-10 E70.3 ICD-9 270.2 OMIM 203100 103470, 203200, 203280, 203290, 203300, 203310, 256710, 278400, 214450, 214500, 220900, 300500, 300600, 300650, 300700, 600501, 604228, 606574, 606952, 607624, 609227 DiseasesDB 318 MedlinePlus 001479 eMedicine derm/12 MeSH D000417 Albinism (from Latin albus, "white"; see extended etymology, also called achromia, achromasia, or achromatosis; not to be confused with albedo) is a form of hypopigmentary congenital disorder, characterized by a partial (in hypomelanism, also known as hypomelanosis) or total (amelanism or amelanosis) lack of melanin pigment in the eyes, skin and hair (or more rarely the eyes alone). Albinism results from inheritance of recessive alleles. The condition is known to affect mammals (including humans), fish, birds, reptiles and amphibians. While the most common term for an organism affected by albinism is "albino" (noun and adjective), the word is sometimes used in derogatory ways towards people; more neutral terms are "albinistic" (adjective) and "person with albinism" (noun). Additional clinical adjectives sometimes used to refer to animals are "albinoid" and "albinic". Albinism is hereditary; it is not an infectious disease and cannot be transmitted through contact, blood transfusions, or other vectors. The principal gene which results in albinism prevents the body from making the usual amounts of the pigment melanin. Most forms of albinism are the result of the biological inheritance of genetically recessive alleles (genes) passed from both parents of an individual, though some rare forms are inherited from only one parent. There are other genetic mutations which are proven to be associated with albinism. All alterations, however, lead to changes in melanin production in the body.[1][2] Albinism was formerly categorized as tyrosinase-positive or -negative. In cases of tyrosinase-positive albinism, the enzyme tyrosinase is present. The melanocytes (pigment cells) are unable to produce melanin for any one of a variety of reasons that do not directly involve the tyrosinase enzyme. In tyrosinase-negative cases, either the tyrosinase enzyme is not produced or a nonfunctional version is produced. This classification has been rendered obsolete by recent research.[3] The chance of offspring with albinism resulting from the pairing of an organism with albinism and one without albinism is low, as discussed in more detail below. However, because organisms can be carriers of genes for albinism without exhibiting any traits, albinistic offspring can be produced by two non-albinistic parents. Albinism usually occurs with equal frequency in both genders.[1] An exception to this is ocular albinism, because it is passed on to offspring through X-linked inheritance. Thus, males more frequently have ocular albinism as they do not have a second X chromosome.[3] Albino Bennett's Wallaby, Bruny Island, Tasmania, Australia Because organisms with albinism have skin that lacks (sufficiently or entirely) the dark pigment melanin, which helps protect the skin from ultraviolet radiation coming from the sun, they can sunburn easily from overexposure. (See human skin color for more information). Lack of melanin in the eye also results in problems with vision, related and unrelated to photosensitivity, which are discussed further below. Most humans and many animals with albinism appear white or very pale; the multiple types of melanin pigment are responsible for brown, black, gray, and some yellow colorations. In some animals, especially albinistic birds and reptiles, ruddy and yellow hues or other colors may be present on the entire body or in patches (as is common among pigeons), due to the presence of other pigments unaffected by albinism such as porphyrins, pteridines and psittacins, as well as carotenoid pigments derived from the diet. Some animals are white or pale due to chromatophore (pigment cell) defects, do not lack melanin production, and have normal eyes; they are referred to as leucistic. The direct opposite of albinism, an unusually high level of melanin pigmentation (and sometimes absence of other types of pigment in species that have more than one), is known as melanism, and results in an appearance darker than non-melanistic specimens from the same genepool.[4] Albinism-like conditions may affect other pigments or pigment-production mechanisms in some animals (e.g. "whiteface", a lack of psittacins that can affect some parrot species.).[5] Another is common in reptiles and amphibians: axanthism, in which xanthophore metabolism, instead of synthesis of melanin, is affected, resuling in reduction or absence of red and yellow pteridine pigments.[6] Of all these conditions, only albinism and melanism affect humans. An albino Wistar rat, a strain commonly used for both biomedical and basic research. The eyes of an animal with albinism occasionally appear red due to the underlying retinal blood vessels showing through where there is not enough pigment to cover them. In humans this is rarely the case, as a human eye is quite large and thus produces enough pigment to lend opacity to the eye, often colouring the iris pale blue. However, there are cases in which the eyes of an albinistic person appear red or purple, depending on the amount of pigment present. The albinistic are generally (but see related disorders below) as healthy as the rest of their species, with growth and development occurring as normal, and albinism by itself does not cause mortality[1] (though the lack of pigment is an elevated risk for skin cancer and other problems.) Many animals with albinism lack their protective camouflage and are unable to conceal themselves from their predators or prey; the survival rate of animals with albinism in the wild is usually quite low.[7][8] However the novelty of albino animals has occasionally led to their protection by groups such as the Albino Squirrel Preservation Society. Intentionally-bred albinistic strains of some animal species are commonly used as model organisms in biomedical study and experimentation. Examples include the BALB/c mouse and Wistar and Sprague Dawley rat strains, while albino rabbits were historically used for Draize toxicity testing. [9] Albino axolotls, zebrafish, medaka and frogs are other common laboratory animals. The yellow mutation in fruit flies is their version of albinism. The incidence of albinism can be artificially increased in fish by exposing the eggs to heavy metals.[10] About 1 in 17,000 human beings has some type of albinism, although up to 1 in 70 is a carrier of albinism genes.[11]

People who have a family history of albinism will be the most likely to get albinism but normal people have a chance of getting it

Albinism is a recessive trait which will result in a lack of pigmentation in the skin eyes and hair. It occurs about 1 in every 17,000 people in the U.S.

In the Philippines,A celebrity that has albinism is redford white.

highly unlikely

Because the chemicals inside the beneficial burn away the organism.

dont go out in the sun, dye all your hair white, and get pink contacts :) (you can get contacts even if you have 2020 vission.

Albinism is a congenital disorder that can be identified with amniocentesis during pregnancy or at birth. They have inherited altered genes that do not make the usual amounts of a pigment called melanin. One person in 17,000 in the U.S.A. has some type of albinism.

Albinism means without pigment. An albino has no melanin in the skin. The hair is also derived from the skin and has no color. The eyes also are derived from the same tissue that form the skin so the iris has no pigment. The eyes appear red because the blood vessels in the back of the eye are what is seen.

Chromosome 9.

Albinos are not blind. They lack the colored pigment that most people have, but their retina works the same as everyone else.

albinism, a hereditary disorder that affects melanin production, there is a lack of pigment in the skin, hair, and eyes. "Memmler's The Human Body in Health and Disease 11th edition"

Yes. Eat blackberries.

People who are color blind can not see some colors or some can see no color. They have a defective gene or genes. These genes are need so the retina can pick up these colors and the brain can interpret them.

No, since "white" people have pigment-producing cells ... they're just set on "low," effectively. True albinos have no pigment in their skin at all.

To quote from an Awake article:

"Albinism, like all other maladies, is a consequence of the imperfection that all humans have inherited from the first man, Adam. (Genesis 3:17-19; Romans 5:12) By means of the ransom sacrifice of Jesus Christ, Jehovah will soon provide perfect health for all those displaying faith. Yes, he is the One "who is healing all your maladies." (Psalm 103:3) Then, albinism will be a thing of the past, for everybody who suffers from it will experience the fulfillment of Job 33:25: "Let his flesh become fresher than in youth; let him return to the days of his youthful vigor."

There are two types of albinism that can be found in a human.

The primary type is oculocutaneous albinism, which is what most people think of when they think of an albino. It can exist in various levels of lack of pigment, from a complete lack of any pigment to a varying degree of that lack. It really isn't 'part' albino, but it is not a partial situation.

A human being can have ocular albinism, which affects only the eyes and not the rest of the body.

For them, yes. Usually people get their Eye color from a ring around the pupil. In Albinos, this ring can be entirely clear. The red is the color of the inside of the Eye shining through. This means that their Eyes take in a lot more light, and can get damaged by that.

They are no danger to anyone else.

same as rest of population

Albinos are just natrally white.

yes

It is a mutation/