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appetite

 
Dictionary: ap·pe·tite   (ăp'ĭ-tīt') pronunciation
n.
  1. An instinctive physical desire, especially one for food or drink.
  2. A strong wish or urge: an appetite for learning.

[Middle English apetit, from Old French, from Latin appetītus, strong desire, from past participle of appetere, to strive after : ad-, ad- + petere, to seek.]

appetitive ap'pe·ti'tive (ăp'ĭ-tī'tĭv, ə-pĕt'ĭ-tĭv) adj.

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Food and Fitness: appetite
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Appetite is a psychological desire to eat and is often related to specific foods. Unlike hunger, it is probably a learned response associated with pleasant tasting and satisfying food. It is an agreeable sensation that aids digestion by stimulating the secretion of saliva and other digestive juices. It also stimulates the desire to eat sufficient food to maintain the body and supply it with enough energy and nutrients to carry out its functions. However, as with other body functions, disorders of appetite occur. Excessive appetite may result in obesity, while diminished appetite is a sign common to many illnesses and may be a manifestation of stress. There is no simple relationship between exercise and appetite, but most experts believe that appetite is likely to increase with exercise only if an individual is normally fairly active and then raises his or her activity even higher. When a sedentary person exercises, appetite may not increase, on the contrary, it may be suppressed (see also anorexia nervosa).

Thesaurus: appetite
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noun

  1. A desire for food or drink: hunger, stomach, taste, thirst. See desire.
  2. A strong wanting of what promises enjoyment or pleasure: appetence, appetency, craving, desire, hunger, itch, longing, lust, thirst, wish, yearning, yen. See desire.
  3. A liking for something: fondness, partiality, preference, relish, taste, weakness. See like/dislike.

Antonyms: appetite
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n

Definition: desire for food, worldly goods
Antonyms: antipathy, aversion, disgust, dislike, distaste, hatred, indifference, loathing, repulsion, revulsion


Dental Dictionary: appetite
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n

A natural or instinctive desire for food.

A psychological desire to eat. Unlike hunger, it is probably a learned response associated with pleasant-tasting and satisfying foods. It is an agreeable sensation undoubtedly necessary for good digestion, and is accompanied by secretion of saliva and digestive juices. It also provides the desire to eat enough food to maintain the body and supply it with sufficient energy to carry out its functions. However, as with other body functions, disorders of appetite occur. Excessive appetite can lead to obesity, while a chronic diminished appetite is a sign of many illnesses and may be a manifestation of stress. See also amphetamines, anorexia nervosa, bulimia.

Appetite is a term implying a strong desire to acquire or participate in, exemplified by terms such as sexual appetite or appetite for life. In the context of food, appetite is used to describe a wanting or liking for particular foods, usually on the basis of their sensory properties (taste and texture) or a psychological attribute (perceived value or symbolic status). In this way appetite is usually distinguished from hunger, which implies a desire or seeking for food arising from a state of need or nutritional deficit. At the beginning of the twenty-first century the understanding of appetite achieves special importance because of its potential role in the worldwide epidemic of obesity, sometimes called a pandemic. Given that, in many parts of the world, people are surrounded by a plentiful supply of food that prevents chronic hunger (though permitting normal meal-to-meal hunger), the capacity to eat food in the absence of hunger or in a low state of hunger assumes special importance. Consequently, understanding appetite and how it can be controlled are urgent tasks in the fight against the obesity epidemic.

Appetite can therefore be defined as a liking for particular foods, or an attraction for foods based on their perceived pleasantness. This is normally referred to as the hedonic dimension of food selection. This characteristic can be described as the subjective pleasure that is derived from the consumption of food; in turn, this can be measured by asking people to rate the magnitude or intensity of pleasure associated with eating or tasting foods. This pleasure arises from the interaction between the person's perceptual capacity (acuity of taste, smell, and sensory feedback from the mouth) and the physical properties of foods. The intensity of the pleasure therefore depends in part on internal (personal) and external (food-related) factors. These food factors can be natural, such as the presence of sweet carbohydrates in fruits or, and much more common now, the deliberate construction of powerful properties in the manufacturing process. It can be hypothesized that the industrial production of foods (designed to possess a combination of properties, for example, sweetness, fattiness, flakiness) has saturated the food supply in many parts of the world with an abundance of appetite-stimulating products. These products include chocolates and desserts, cheese, meat, and pastry combinations, and many types of fried snacks. The inherent attractiveness of such products can stimulate eating in the absence of any obvious need for nutrients.

Biological Basis of Appetite

Is there a biological basis for appetite and for the degree of attractiveness of specific types of foods? It does seem that human beings derive pleasure from particular food properties—the qualities of sweetness and fattiness are prominent. It is generally understood that, during the course of human evolution, a preference for foods with these properties would lead people to consume foods that possessed energy—yielding value, for example, the nutritional value of carbohydrates and the energy value of fats. Consequently the value of these traits for survival has almost certainly persisted until the present day, at which stage these genetic dispositions may be detrimental in the current "obesigenic" environment (but useful when foods with these properties were scarce). The word "obesigenic" was coined around the end of the twentieth century to suggest an environment that promoted weight gain through the abundance, attractiveness, and marketing of food consumption, together with reduced opportunities for physical activity. It is recognized that most cultures contain highly prized food habits based on foods that are either sweet or fatty, and sometimes a combination of both—when the palatability can be intense.

Do these genetic traits based on the pleasurable qualities of food have a basis in brain processes? The intrinsic sensory attractiveness of food is mediated by "reward" pathways in the brain. These pathways promote various types of pleasure and can be artificially stimulated by drugs. By using drugs as tools it has been found that particular neurochemical transmitters are involved in the process of reward; these transmitters include dopamine, opioid, and cannabinoid molecules together with their specific receptors. It can also be demonstrated empirically that the areas of the brain that subserve the most intense pleasure can be stimulated both by food sensations (arising from sensory pathways), and modulated by signals of need (arising from the body's energy stores). This means that a nutritional deficit, indicated by a low body weight, for example, can sensitize the reward system so as to increase the measured pleasantness of foods. In practice this would mean that a person who had been coerced into losing substantial body weight would display an increased rating of pleasantness for certain foods. This can be seen as a useful biological mechanism and is given credibility via the long-known phenomenon called alliesthesia, which is perceiving an external stimulus as pleasant or not, depending on internal stimuli. This concept is based on the biological notion of pleasure being a useful trait.

However, it seems certain that another mechanism must also be at work. This mechanism is based on the recognition that some people who gain weight easily and become obese possess traits that lead them to derive a high degree of pleasure from food. Consequently, foods with potent sensory properties are attractive targets for such people and this increasing pleasantness can lead to overconsumption and weight gain. There is clear evidence that obese women rate sweet/fatty foods very highly and consume substantial quantities; other studies have shown that obese people show preferences for fatty foods and for the taste of fat. After eating, obese subjects frequently rate the same food as being more pleasant than do lean subjects. In this way the expression of appetite—a heightened pleasure of eating—can be seen to contribute to increasing body weight and obesity. This arises from endogenous traits to derive pleasure from food (sometimes specific foods) in conjunction with an abundance of foods possessing a profile of pleasure-stimulating properties.

Hedonics and Hunger

The identification of the pleasure response of appetite with a neurochemical substrate also helps to differentiate the hedonic dimension of food from the hunger dimension. The term "hedonic," derived from the Greek word hedone, refers to the seeking of pleasure. Experimental studies in human subjects have shown that a drug called naloxone that blocks opioid receptors can reduce the perceived pleasantness of food without diminishing hunger. In contrast a drug such as fenfluramine, that acts upon serotonin receptors, can substantially reduce hunger without changing the perceived pleasantness of food. This type of pharmacological dissection indicates that the overall control of food intake depends upon both appetite (signals of pleasure) and hunger (signals of absence of food). However, the separation of the systems is not complete since experimental investigations have shown that while people are eating very good-tasting food their hunger is elevated. This elevation serves to prolong the meal so that more food is consumed. The relationship is, however, asymmetrical: although pleasantness increases hunger, a state of satiety does not reduce the perception of pleasure. Indeed, even when people report feeling full, a very palatable food can often still be eaten. Surveys have shown that foods rated as most palatable (pleasant) are associated with the largest meal sizes and with the greatest amount of food consumed. The results of scientific observations are therefore consistent with the widespread belief that people eat more of good-tasting food. However, people do not always choose to eat the most delectable or most pleasure-giving food; eating also depends on the appropriateness of the food for the particular social context. This is a good example of a cultural rule overcoming a biological response. However, much overeating in certain cultures arises from the strength of the biological response or perceived intensity of pleasure overcoming the cultural convention.

The Palatability Dilemma

The potency of appetite in stimulating food consumption creates a problem in the present climate of escalating levels of obesity. There are now strong intentions in the nutraceutical sector of the food industry to encourage the production of functional foods for appetite control; that is, foods that possess satiety-inducing or hunger-suppressing properties. The word "nutraceutical" was coined to resemble the word "pharmaceutical" and refers to those foods that may have specific functional effects generated in a manner similar to the way in which drugs work. However, advances in food technology have, over the years, been able to bring about an increase in the overall pleasantness (palatability) of foods entering the market-place. Indeed, one of the legitimate goals of the food industry is to make eating a source of pleasure. It can be observed that for many people, eating is the cheapest form of pleasure available on a day-to-day basis. However, improving palatability means increasing the sensory attractiveness of foods and the willingness of people to consume such foods. Satiety implies reducing the willingness of people to consume. The question therefore arises whether it is possible for the food industry to increase the palatability of foods without weakening satiety—and vice versa. The balance between palatability and satiety is the essence of the interaction between hunger and hedonics in the control of food intake. It remains to be determined how the interplay between these factors contributes to the current obesity epidemic.

Bibliography

Blundell, John E., and Peter J. Rogers. "Hunger, Hedonics and the Control of Satiation and Satiety." In Chemical Senses, edited by Mark I. Friedman and Michael G. Tordoff. New York: M. Dekker, 1991.

de Castro, J. M., F. Bellisle, and A.-M. Dalix. "Palatability and Intake Relationships in Free-Living Humans: Measurement and Characterization in the French." Physiology and Behaviour 68 (2000): 271-277

Mela, D. J., and Peter J. Rogers. Food, Eating and Obesity: ThePsychoBiological Basis of Appetite and Weight Control. London: Chapman and Hall, 1998.

Mela, D. J., and D. A. Sacchetti. "Sensory Preferences for Fats: Relationships with Diet and Body Composition." American Journal of Clinical Nutrition 53 (1991): 908-915.

Yeomans, M. R. "Taste, Palatability and the Control of Appetite." Proceedings of the Nutrition Society 57 (1998): 609–615.

—John E. Blundell; Joanna Le Noury

Veterinary Dictionary: appetite
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The desire for food. It is stimulated by the sight, smell or thought of food and accompanied by the flow of saliva in the mouth and gastric juice in the stomach. Appetite is psychological, dependent on memory and associations, as compared with hunger, which is physiologically aroused by the body's need for food. Its existence in animals can only be conjectured on the response in the form of food intake. Chronic loss of appetite is known as anorexia.

  • decreased a. — see anorexia.
  • depraved a. — see pica. Called also allotriophagia.
  • increased a. — see hyperorexia, polyphagia.
  • salt a. — the appetite for salt displayed by animals, especially ruminants, at pasture. Used in the manangement of cattle on extensive range. See also salting.
  • a. stimulants — there are several methods available. Bitters such as gentian and pulv. nux. vomica exert some effect in ruminants, probably by stimulating alimentary tract mucosa. Anabolic steroids are sometimes used for this purpose in dogs and cats. A nutritional deficiency of zinc or the B vitamin complex depresses acuity of appetite and dietary supplementation with it can be effective as an appetite stimulant. Suppression of the medullary satiety center by barbiturates and benzodiazepines is also recommended. There are no well-regarded stimulants of the hunger center of the lateral hypothalamus.
Devil's Dictionary: appetite
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A cynical view of the world by Ambrose Bierce


n.

An instinct thoughtfully implanted by Providence as a solution to the labor question.


Word Tutor: appetite
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pronunciation

IN BRIEF: A desire or wish for food.

pronunciation When I feel carsick, I have absolutely no appetite.

Tutor's tip: No matter how big your "appetite" (hunger) is, you still can't eat a mineral like "apatite."

Quotes About: Appetite
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Quotes:

"The most violent appetites in all creatures are lust and hunger; the first is a perpetual call upon them to propagate their kind, the latter to preserve themselves." - Joseph Addison

"Appetite is essentially insatiable, and where it operates as a criterion of both action and enjoyment (that is, everywhere in the Western world since the sixteenth century) it will infallibly discover congenial agencies (mechanical and political) of expression." - Marshall Mcluhan

Wikipedia: Appetite
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The appetite is the desire to eat food, felt as hunger. Appetite exists in all higher life-forms, and serves to regulate adequate energy intake to maintain metabolic needs. It is regulated by a close interplay between the digestive tract, adipose tissue and the brain. Decreased desire to eat is termed anorexia, while polyphagia (or "hyperphagia") is increased eating. Dysregulation of appetite contributes to anorexia nervosa, bulimia nervosa, cachexia, overeating, and binge eating disorder.

Contents

Regulation

The regulation of appetite has been the subject of much research in the last decade. Breakthroughs included the discovery, in 1994, of leptin, a hormone that appeared to provide negative feedback. Later studies showed that appetite regulation is an immensely complex process involving the gastrointestinal tract, many hormones, and both the central and autonomic nervous systems.

Effector

The hypothalamus, a part of the brain, is the main regulatory organ for human appetite. The neurons that regulate appetite appear to be mainly serotonergic, although neuropeptide Y (NPY) and Agouti-related peptide (AGRP) also play a vital role. Hypothalamocortical and hypothalamolimbic projections contribute to the awareness of hunger, and the somatic processes controlled by the hypothalamus include vagal tone (the activity of the parasympathetic autonomic nervous system), stimulation of the thyroid (thyroxine regulates the metabolic rate), the hypothalamic-pituitary-adrenal axis and a large number of other mechanisms. Opioid receptor-related processes in the nucleus accumbens and ventral pallidum effect the palatability of foods.[1]

Sensor

The hypothalamus senses external stimuli mainly through a number of hormones such as leptin, ghrelin, PYY 3-36, orexin and cholecystokinin; all modify the hypothalamic response. They are produced by the digestive tract and by adipose tissue (leptin). Systemic mediators, such as tumor necrosis factor-alpha (TNFα), interleukins 1 and 6 and corticotropin-releasing hormone (CRH) influence appetite negatively; this mechanism explains why ill people often eat less.

In addition, the biological clock (which is regulated by the hypothalamus) modifies hunger. Processes from other cerebral loci, such as from the limbic system and the cerebral cortex, project on the hypothalamus and modify appetite. This explains why in clinical depression and stress, energy intake can change quite drastically.

Role in disease

A limited or excessive appetite is not necessarily pathological. Abnormal appetite could be defined as eating habits causing malnutrition and related conditions such as obesity and its related problems.

Both genetic and environmental factors may regulate appetite, and abnormalities in either may lead to abnormal appetite. Poor appetite (anorexia) may have numerous causes, but may be a result of physical (infectious, autoimmune or malignant disease) or psychological (stress, mental disorders) factors. Likewise, hyperphagia (excessive eating) may be a result of hormonal imbalances, mental disorders (e.g. depression) and others.

Dysregulation of appetite lies at the root of anorexia nervosa, bulimia nervosa and binge eating disorder. In addition, decreased response to satiety may promote development of obesity.

Various hereditary forms of obesity have been traced to defects in hypothalamic signalling (such as the leptin receptor and the MC-4 receptor), or are still awaiting characterisation (Prader-Willi syndrome).

Pharmacology

Mechanisms controlling appetite are a potential target for weight loss drugs. Early anorectics were fenfluramine and phentermine. A more recent addition is sibutramine which increases serotonin and noradrenaline levels in the central nervous system. In addition, recent reports on recombinant PYY 3-36 suggest that this agent may contribute to weight loss by suppressing appetite.

Given the epidemic proportions of obesity in the Western world, developments in this area are expected to snowball in the near future, as dieting alone is ineffective in most obese adults.

References

  1. ^ Wassum KM, Ostlund SB, Maidment NT, Balleine BW. (2009). Distinct opioid circuits determine the palatability and the desirability of rewarding events. Proc Natl Acad Sci U S A. 106:12512–12517 PMID 19597155 doi:10.1073/pnas.0905874106
  • Neary NM, Goldstone AP, Bloom SR. Appetite regulation: from the gut to the hypothalamus. Clin Endocrinol (Oxford) 2004;60:153-60. PMID 14725674.
  • Wynne K, Stanley S, Bloom S. The gut and regulation of body weight. J Clin Endocrinol Metab 2004;89:2576–82. PMID 15181026.

See also


Translations: Appetite
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Dansk (Danish)
n. - appetit

idioms:

  • appetite for knowledge    videbegærlig

Nederlands (Dutch)
eetlust, begeerte

Français (French)
n. - appétit, (fig) goût

idioms:

  • appetite for knowledge    (avoir) soif de connaissance

Deutsch (German)
n. - Appetit, Verlangen

idioms:

  • appetite for knowledge    Wißbegier

Ελληνική (Greek)
n. - όρεξη, (ψυχολ.) ορμή

idioms:

  • appetite for knowledge    όρεξη για μάθηση

Italiano (Italian)
appetito

Português (Portuguese)
n. - apetite (m), desejo (m), ânsia (f)

idioms:

  • appetite for knowledge    fome (f) de conhecimento
  • have an appetite    ter apetite
  • ravenous appetite    apetite exagerado
  • voracious appetite    apetite (m) voraz

Русский (Russian)
аппетит

idioms:

  • appetite for knowledge    жажда знаний
  • have an appetite    иметь хороший аппетит
  • ravenous appetite    зверский аппетит
  • voracious appetite    зверский аппетит

Español (Spanish)
n. - apetito, apetencia

idioms:

  • appetite for knowledge    ansia de saber, afán de conocimientos

Svenska (Swedish)
n. - aptit, begär

中文(简体)(Chinese (Simplified))
爱好, 食欲, 欲望

idioms:

  • appetite for knowledge    对知识的渴求热爱

中文(繁體)(Chinese (Traditional))
n. - 愛好, 食欲, 欲望

idioms:

  • appetite for knowledge    對知識的渴求熱愛

한국어 (Korean)
n. - 식욕, 욕망

日本語 (Japanese)
n. - 食欲, 欲求, 欲望

idioms:

  • appetite for knowledge    知識欲
  • have an appetite    食欲がある

العربيه (Arabic)
‏(الاسم) شهيه, شهوه, ميل فطري أو مكتسب‏

עברית (Hebrew)
n. - ‮תיאבון, חשק‬


 
 

 

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