I have not seen it myself, but it is the belief of some people in natural health that it can be a trigger through molecular mimicry. Although, I have been looking for studies on it. (I was looking for such studies, when I found this question). It may be that such studies have not been done, but that doesn't mean practitioners clinical experience is useless. I would add, that the triggering of auto-immune disease through molecular mimicry in general is more established (at least in research, although conventional medicine doesn't care about it now). Poor GI health, IBS, dysbisosis and low secretory IgA is common in auto-immune disease and should be corrected as a possible underlining trigger. If non-ai hypothyroidism, this should still be corrected as a stress on the body. Either case, yes, it may be implicated, as the body may adapt to increase stress by lowering metabolic rate (and hence - thyroid hormone).
Saul Marcus
drsaulmarcus.com
Hypothyroidism causes you to retain water that leads to bloating.
Hypothyroidism does not cause mental "retardation". MR is caused by genetics usually. Hypothyroidism is treatable.
The child was infected with parasite cause by amoeba.
Removal of the thyroid gland because of cancer or other thyroid disorders can result in hypothyroidism.
Infections that depress thyroid hormone production usually cause permanent hypothyroidism.
Freshwater amoeba in salt water will have a higher solute content outside of the amoeba. The water in the amoeba will want to move out of the amoeba and into the environment. This will cause the amoeba to shrivel and die.
yes
dysentery
Amoebic dysentery, for one.
No. An overproduction of TSH would cause hyperthyroidism.
There are several types of amoeba, including the common amoeba called Amoeba proteus, which can be found in freshwater environments. Other types of amoeba include Entamoeba histolytica, which is a parasite that can cause amoebic dysentery, and Naegleria fowleri, a pathogenic amoeba that can cause a rare and fatal brain infection called primary amoebic meningoencephalitis.
Some patients report weight gain with subclinical hypothyroidism. Although the T4 is normal in subclinical hypothyroidism by definition, some patients report symptoms with only the elevated TSH.