i really dont know
Individual pathogens can develop resistance to antimicrobial drugs through several mechanisms. These include acquiring genes that code for drug resistance, mutations that render the drug targets less susceptible to the drugs, and the ability to pump out or destroy the drugs. These adaptations occur through genetic changes that give the pathogens a survival advantage and allow them to evade the action of the drugs.
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Fungal infection present special problems in term of treatment for several reasons. They tend to develop clinical resistance, microbial resistance, and emergence of new pathogens.
resistance mechanisms often carried by plasmids can be easily transmitted in bacterial populations by conjugation
A sperm or egg alone can not develop into a new individual. Only an egg fertilized by a sperm does.
it inherited the allele that made it resistant
They are said to be susceptible.
Immunity
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When connected in series, the overall effective resistance of a bunch of individual resistors is the sum of the individual resistances. It's always more than the resistance of any individual. When connected in parallel, the reciprocal of the overall resistance of a bunch of individual resistors is the sum of the reciprocals of the individual resistances. It's always less than the resistance of any individual. When two resistors are connected in parallel, the overall effective resistance of the pair is (the product of the two individual resistances) divided by (the sum of the two individual resistances). It's always less than the smaller individual resistance.
It depends on the values of the individual resistors. But if each resistor is identical, then the total resistance will be one-quarter that of an individual resistor.
Carrier
Those pathogens that survive the drug attack and then reproduce progeny having this same resistance is a classic selection scenario.
Add all the individual resistance values.
It is an acquired immunity
The resistance of a series circuit is simply the sum of the individual resistors.
Fungal infection present special problems in term of treatment for several reasons. They tend to develop clinical resistance, microbial resistance, and emergence of new pathogens.
I observe that the total effective resistance of several resistors in series is the sum of the individual resistance values of the individual resistors.