How does physical and occupational therapy treat myasthenia gravis?
Physical and occupational therapists provide strategies to maintian daily activities. Shower stools, rolling carts for shopping and exercises to maintain posture all help avoid fatigue.
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Prevalence of MG in the United States . The prevalence of MG in the United States is estimated to be about 20/100,000 population. However, MG is probably under diagnosed and the prevalence may be higher. Myasthenia Gravis occurs in all races, both genders, and at any age. MG is not thought to be …directly inherited nor is it contagious. It does occasionally occur in more than one member of the same family. \n. \nSource: Myasthenia Foundation of America, Inc. (MGFA)\nhttp://www.myasthenia.org/amg_whatismg.cfm\nTaken From Article entitled: What is Myasthenia Gravis (MG) ( Full Answer )
Answer . It is an autoimmune neuromuscular disease, characterized by muscle weakness and fatiguability. Where antibodies are directed against the muscle's own acetylcholine receptors that stimulates its contraction. Leading to increasing weakness of the body muscles during periods of activity; s…ince the muscle contracts less at every stimulus. MG is a chronic autoimmune neuromuscular disease characterized by degrees of weakness of the skeletal muscles of the body which increases during periods of activity and improves after periods of rest. ( Full Answer )
Answer . Occupational Therapy is treatment that helps individuals achieve independence and improve one's ability to perform daily activities... Physical therapists focus on the large motor groups that contribute to walking, reaching, standing and physical activities. They work on strength, bala…nce, range of motion and swelling as well as pain to encourage independence.. PT's and OT's work in tandem for the best outcome. ( Full Answer )
Answer . Both myasthenia gravis (MG) and Graves disease (GD) are autoimmune diseases. Myasthenia gravis is characterized by impaired transmission of the neural stimuli to the muscles due to circulating antibodies anti-acetylcholine receptor. Graves disease is autoimmune hyperthyroidism due to ant…ibodies anti-TSH (Thyroid Stimulating Hormone) receptor. The prevalence of auoimmune thyroid diseases in MG is about 5-10%. The incidence of MG in GD is fairly low: aprox 0,2%. One problem is the overlapping of the symptoms which may cause diagnostic confusion. Not only clinical manifestation but also some morphological aspects, like thymus hyperplasia confirmed by thoracic CT are shared by the 2 diseases. Generally, only mild forms of MG (especially ocualr form) are associateed with GD. When in fact it was a myasthenia-like syndrome as a manifestation of GD remission of GD will cure MG-like symptoms also. ( Full Answer )
As they address two different areas (occupational therapy- the tasks of living, physical therapy- functioning as it relates to mobility)- there is no quantifiable answer to that question.
I am a Physical Therapist. MG pts. do not realize the rapidity of which there muscles fatigue during exercise... With proper timing of exercise repititions (usually slow to moderate speed),and proper intervals of rest (recuperation between exercises -usually 3 to 5 minutes) MG patients can successfu…lly exercise without undue fatigue . Each patient is different and should consult with a licensed and skilled Physical Therapist before and during any rehabilitation program ( Full Answer )
I believe that my exposure to agent orange in Vietnam in 1968-1969 caused my myasthenia gravis...I am trying to find some supporting information on this...My e-mail address is egreene42@AOL.com .thanks for your help...
Physical therapy focuses on movement while occupational therapy focuses on function and participation in activities of daily living
Yes its harder and and more school. PT - need doctorate degree. OT - need masters degree. The classmates i had that couldn't get the grades in PT school often switched to OT and were able to pass.
The underlying cause is not yet known, but it has not been linked to hereditary disease. It is an autoimmune disease. In these disorders, the body's immune system starts to attack itself, mistaking its tissues as foreign. See the related link below for more about this neuro-muscular autoimmune disea…se. ( Full Answer )
I have had MG since 2000. As a result of long term medication I getbad lower back pain, also brought on when my MG is playing up. Ihave just brought a tens machine, but am worried that it may affectmy MG, since it is pretty much under control, due to medication.Many thanks.
these drugs improve the nerve signals to the muscles therefore there will be an increase in the muscle strength.
They're both very important fields and are equally needed. Physical therapy deals with rehabilitation of bones, joints, and muscles and gross motor skills. Occupational therapy deals more with the thought processes and coordination of fine motor skills. A stroke victim that has experienced loss of b…rain function and paralysis might require both physical and occupational therapy as an example. We also live in a fairly active society, one where sports injuries and accidents are common. Unfortunately, or rather fortunately for you if you're thinking of this as a career path, both types of therapists will be needed for many years to come. ( Full Answer )
Initial symptoms of MG may include difficulty speaking (dysarthria), difficulty swallowing (dysphagia), drooping eyelids (ptosis), and double vision (diplopia). Patients often have nasal-sounding speech and weak neck muscles that give the head a tendency to fall forward or backward.These symptoms oc…cur in about 90% of MG cases, are usually intermittent (i.e., come and go), and may disappear for weeks and then recur. Generalized weakness often develops in the trunk, arms, and legs within a year of onset. Arm muscles usually are affected most severely. Muscle weakness tends to worsen as the day progresses, especially after prolonged activity. Pregnancy can improve, worsen, or have little effect on MG symptoms. Frequently, symptoms first occur during pregnancy or after delivery. ( Full Answer )
People with MG suffer with debilitating muscle weaknesses in their arms, legs, face, throat, hands and eyes. The symptoms of muscle weakness may occur at any time but most often after normal use of the extremities, i.e. Taking a walk may cause leg weakness and reading a book may cause eye weakness
Myasthenia Gravis is an autoimmune disease of the neuromuscular system that is characterized by high plasma levels of acetylcholine receptor (AChR) antibodies, which invoke an immune response against these receptors. Production of these antibodies are caused by molecular mimicry of a self antigen. T…-helper cells, a type of immune system cells, can come across antigen presenting cells that display an antigen that is similar, in sequence, to AChR's. The T-helper cells are activated in this manner and can cause an immune response (antibody production) against AChR's. In essence, the Herpes virus is known to have a similar sequence to AChR's, therefore, the existance of the Herpes virus increases the risk of Myasthenia Gravis development. ( Full Answer )
I've had MG (diagnosed) for 17 years. It would seem that MG could result in gastroparesis. However, it is possible that MG treated with large doses of prednisone or other corticosteroids over a long time might be the route. The prednisone causes a challenge to blood sugar control, sometimes leadin…g to diabetes in the MG patient. Diabetes is said to be the leading known cause of gastroparesis. Be very careful weaning off of prednisone; follow a knowledgeable MD's instructions. It is my experience that the AMA physicians can be very helpful with MG, as can naturopaths, accupunturists, etc. when working in cooperation with an MD. None, however, including the MDs, know everything about MG - particularly its expression and course in atypical patients. ( Full Answer )
Although polyvinyl chloride (PVC) is associated with some healtheffects, through Phthalates, they are usually either breast cancerand endocrine disruptions. Myasthenia Gravis is when theneurotransmitter acetylcholine is blocked in the eye, causing eyemuscles to become relaxed and either partially or… completelyuncontrollable. It is unlikely that PVC cause myasthenia gravis. ( Full Answer )
Corticosteroids provide significant improvement or complete relief of symptoms in more than 75% of people, and some improvement occurs in most of the rest.
The need for the drug varies from day to day and during the same day in response to infection, menstruation, emotional stress, and hot weather. Different muscles respond differently.
Immune globulin given intravenously results in improvement in more than half of MG patients, usually beginning within one week of therapy and lasting for several weeks or months.
Plasma exchange is used as a short-term intervention for patients with sudden worsening of symptoms, to rapidly improve strength before surgery, and as a chronic intermittent treatment.
Thymectomy or thymus removal is recommended for most with this disease. Greatest benefit occurs 2 to 5 years afterwards. The best response are in young people early in the onset.
It takes 6 to 8 weeks of drug therapy to see improvement. About 1/3 of patients become weaker temporarily after starting prednisone, usually in the first 7 to 10 days, but this weakness is temporary.
Within a year of onset, about 85-90% of cases develop generalized MG, with weakness in the trunk, arms, and legs. About 10-15% have weakness only eye muscles. Onset may be sudden.
Study of CellCept and prednisone as immunotherapy in the treatment of MG. Another is the study of Etanercept to determine if it improves muscle strength in patients with MG.
What are the possible adverse side effects associated with taking cyclosporine to treat myasthenia gravis?
Possible side effects include drug contraindications that interfere with cyclosporine metabolism and life threatening infections are a risk.
The major disadvantages of chronic corticosteroid therapy are the side effects, such as weight gain and fluid retention.
Congenital MG is an inherited condition caused by a genetic defect. The medical condition develops at or shortly after birth and causes generalized symptoms.
The prevalence of MG in the United States is estimated to be 14 per 100,000 population, which equals approximately 36,000 cases. However, this disease is probably under diagnosed.
Symptoms usually progress to maximum severity within 3 years. With treatment, the outlook for most patients with MG is bright: significant improvement of muscle weakness and nearly normal lives.
MG treatments are practical rather than curative and include cholinesterase inhibitors, thymectomy, corticosteroids, immunosuppressant drugs, plasma exchange, intravenous immune globulin.
Persons with disease onset after the age of 60 and those with thymomas (tumor on the thymus) do not respond well to thymectomy.
Myasthenia gravis occurs in all ethnic groups and both genders. Initial studies showed women are more often affected than men but as the population ages men are more affected than women.
10 percent of patients have a tumor in the thymus, that is usually benign, and 70% have changes that indicate an active immune response.
Muscles that control eye, eyelid movements, facial expression, chewing, talking, swallowing are often affected. The muscles that control breathing and neck and limb movements may also be involved.
The physical therapist can provide noninvasive therapies, such as ultrasound or diathermy to project heat deep into the tissues of the back or administer manual therapy, if mobility of the spine is impaired.
affects the neuromuscular junction, interrupting the communication between nerve and muscle, and thereby causing weakness. A person with MG may have difficulty moving their eyes, walking, speaking clearly, swallowing
It is characterized by fatigue and muscle weakness that at first may be confined to certain muscle groups, but then may progress to the point of paralysis
MG affects "voluntary" muscles, which are those muscles under conscious control responsible for movement. It does not affect heart muscle or the "smooth" muscle found in the digestive system and other internal organs
Myesthenia Gravis can be controlled and may go to Remission for along time for about 50% of the patients. With medications thatincrease muscle strength and with subsequent removal of the Thymus(usually abnormal in patients with MG), there is a fairly goodprognosis to eliminate symptomps that may lea…d to remission of thedisease. But there is still a percentage of it coming back. So no,there is no Cure but it can be managed. ( Full Answer )
The patient will also undergo physical and occupational therapy to keep the burned areas from becoming inflexible and to minimize scarring.
An occupational therapist can suggest home or work modifications to reduce fatigue and improve function.
Physical therapy can help relieve secondary consequences of torticollis. Regular muscle stretching prevents contracture, or permanent muscle shortening. Pain and spasm may be temporarily lessened with application of heat or ice.
No it can't be cured but they are still doing studies to see if there is a cure
skeletal (voluntary) muscle weakness. . eyes : drooping eyelids(ptosis) and blurred vision. . mouth : difficulty in chewing, speaking, or swallowing . arms, hands, finger : difficult to perform simple manual tasks. . leg : standing or walking difficult. . respiratory muscles : breathing proble…m . dysphonia. The unique features of myasthenia gravis are fatigability,fluctuation of function, and restoration of function after re ( Full Answer )
The severity ranges depending on how long the patient has been living with the condition. Certain medications may be used to treat symptoms and slow down the disease progression.
Being a therapy aide You could make 25000$ a year. And it is not a regulated profession so You can be both physical and occupational therapy aide.
Aminoglycosides inhibit the release of acetylcholine from the motor nerve. Myestenia gravis patients are more susceptible to this effect. So these drugs are to be avoided in myestenia gravis patients.
Neostigmine is a competitive irreversible inhibitor ofAcetylcholinesterase (AChE), an enzyme responsible for breakingdown acetylcholine (ACh). Myasthenia gravis is caused by the bodyproducing too little ACh receptors. As stimulating the AChreceptors is needed for nervous transmission, the nerve sign…alscannot be transmitted causing muscle weakness and fatigue.Neostigmine is hence used to inhibit AChE so that less ACh getsbroken down. This leads to more ACh binding to the ACh receptorscausing muscular contraction. ( Full Answer )