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1. Carbon monoxide binds to hemoglobin, preventing the affected RBC's to carry a full load of oxygen. So, the oxygen to tissues decreases, which will stimulate the kidneys to synthesize and release more erythropoietin. This hormone increases RBC production in an effort to get more oxygen to the tissues. Thus, the individual may have polycethemia (an abnormal excess of RBC's) and high blood viscosity. Viscous blood increases a person's chance for stroke and heart failure.

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Q: How does the body compensate sublethal effect of carbon monoxide in smokers?
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