Yes
no
IgG and IgM can fix complement by activating the C3 protein at the end of the complement cascade allowing ADCC (antibody dependent cell- mediated cytoxicity) to occur. I have never heard of an IgC antibody. In humans there are only 5 classes of antibodies: IgM, IgG, IgA. IgD & IgE.
This statement is not accurate. Pathogens can provoke both cell-mediated and antibody-mediated responses at the same time. The type of immune response generated depends on the specific characteristics of the pathogen and the immune system of the host.
active active B for plato users
It is also called DTH(delayed type hypersensitivity) is cell mediated instead of antibody
T cell mediated immunity and B cell immunity (Antibody secretion.
supressor t cells
Membrane cofactor protein (MCP) acts as a cofactor for Factor I, which degrades C3b and C4b proteins, thus preventing the amplification of complement activation. By accelerating the decay of C3 convertases, MCP helps regulate the complement system and protect host cells from excessive complement-mediated damage.
Lymphocyteononuclear white blood cell that is responsible for humoral (antibody mediated) and cell mediated immunity.Lymphs Electronic is a lab test to measure the amount of this kind of white blood cell. The results are reported in percentages.There is also a Lymphs Absolute test and results are reported in K/uL
Epidermal cells that aid in the immune response include melanocytes.
The basic principles are; antibodies will be produced in any signs of infection, or abnormality. Then these antibodies are desiged specifically to target, and bind to specfic antigens.
Receptor-mediated exocytosis of glucose refers to the process where specialized receptors on the cell surface recognize glucose molecules and trigger the release of glucose-containing vesicles from within the cell to the outside environment. This mechanism allows cells to regulate the uptake and release of glucose in response to specific signals or conditions.