That is one of the theories. The other theory is tied to the function of dopamine.
Dopamine inhibits another chemical called GABA. Without any GABA dopamine doesn't really do anything.
The other theory is that they have normal levels of Dopamine and low levels of GABA.
These theories came about by the observation that anti-dopamine drugs help the symptoms.
The simple answer is we're not quite sure yet.
Not true .
Excess dopamine is believed to play a significant role in the development of schizophrenia, particularly in relation to the disorder's positive symptoms, such as hallucinations and delusions. The dopamine hypothesis suggests that overactivity in certain dopamine pathways in the brain contributes to these symptoms. However, schizophrenia is a complex disorder, and factors such as genetics, environment, and other neurotransmitter systems also contribute to its onset and progression. Thus, while excess dopamine is a key factor, it is not the sole cause of schizophrenia.
Excess levels of dopamine, a neurotransmitter, is associated with the development of psychosis. This neurotransmitter plays a key role in regulating mood, attention, and motivation, and abnormalities in dopamine levels can lead to symptoms of psychosis such as hallucinations and delusions.
Schizophrenics Anonymous was created in 1985.
The dopamine hypothesis of schizophrenia suggests that an overactive dopamine system in the brain contributes to the symptoms of schizophrenia. Specifically, an excess of dopamine activity in certain areas of the brain is believed to be a factor in the development of psychosis in schizophrenia.
As far as we know, dopamine is the main culprit in psychosis. Autospies have shown excess dopaminergic receptors in the brain's of people who suffered psychosis. Other neurotransmitters are also affected.
A lack of dopamine causes Parkinson's disease. As dopamine is an interneuron synaptic neurotransmitter, it controls the passage of impulses along different neurons. If it can't regulate the signal, this leads to frequent involuntary movements as in Parkinson's.
Mesolimbic pathway - true. It travels from the ventral tegmental area to the nucleus accumbens.
True. Cocaine causes the sudden release in the brain of a neurotransmitter called dopamine. Source. Prentice Hall
Dopamine overactivity is most clearly related to conditions such as schizophrenia, where an excess of dopamine activity in certain brain regions is believed to contribute to symptoms like hallucinations and delusions. It is also associated with disorders like bipolar disorder and attention deficit hyperactivity disorder (ADHD) where dopamine dysregulation is believed to play a role in the manifestation of symptoms.
Amrita Bagga has written: 'Dermatoglyphics of schizophrenics' -- subject(s): Dermatoglyphics, Physiology, Schizophrenics
They are postulated to be 1) excess secretion of dopamine, 2) deficiency of serotonin, and 3) deficiency of norepinephrine (among others).