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Q: Is stomach acid a histamine
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A powerful stimulant of acid secretion in the stomach is?

Histamine


What increases acid secretion in the stomach blocked by cimetidine?

Histamine


How does ranitidine work?

Ranitidine (Zantac) is an H2 blocker. Histamine is released by nerves to the stomach and bind to H2 receptors to signal the stomach to make acid. Ranitidine blocks this interactions and so decreases acid production. The drugs called "antihistamines" block the H1 histamine receptors so that histamine (the same chemical) released by immune cells can't interact with other immune cells to cause allergies.


Is ranitidine an antacid?

Ranitidine is an anti acid, used for people with gastroparesis or stomach ulcer, also used for acid reflux patience. If by antacid you mean the tablets people chew to neutralize too much stomach acid, like Tums or Rolaids, it is not that kind of antacid. Instead of neutralizing the acid that's already there, it prevents your stomach from making so much acid. Here is a description of how it works, from a medical website: Ranitidine acts in the stomach to decrease the production of stomach acid. It works by blocking histamine H2 receptors that are found on the cells in the stomach lining. A natural body chemical called histamine normally binds to these receptors, causing the cells to produce stomach acid. By blocking the H2 receptors, ranitidine prevents histamine from binding to them. This stops the cells from producing stomach acid.


Why are histamine H2 antagonists recommended for gastroesophageal reflux?

they block acid production of the stomach reducing the acidity


Why are histamine antagonists recommended for heart burn?

Histamine receptors in the stomach control acid production, and blocking them reduces acidity. The H2 receptors in the stomach are treated with different agents than the H1 receptors in the brain, there is little overlap.


Factors controlling secretion of the stomach?

The parietal cell in the stomach secrets gastric acid, which is hydrochloric acid (HCl). The Secretion of HCl by the parietal cell is stimulated by receptors for acetylcholine (muscarinic), histamine, and gastrin. The enterochromaffin cells (ECL) also have gastrin receptors and muscarinic receptors. ECL cells release histamine which reacts with H2 receptors (histamine 2 receptors) on parietal cells. Somatostatin is released by delta cells and decreases histamine release by ECL cells.


What are H-2 blockers?

Histamine H-2 receptor blockers act by stopping the pathway that leads to the secretion of stomach acid.


Why do peptic ulcers often develop in individuals with mastocytosis?

Peptic ulcers are frequent because of the increased stomach acid stimulated by histamine.


Which dietary mineral is necessary for producing stomach acid for protein digestion?

Zinc, sodium chloride, and certain b vitamins for adequate histamine release.


Is stomach acid acid or alkali's?

stomach acid is and acid


Is stomach acid alkalis or acid?

stomach acid is and acid