Ca2+
calcium - Ca2+
The cause of synaptic delay is attributed mainly to the time needed for the synaptic vesicles to release neurotransmitter into the synaptic cleft. While it can be considered a combination of binding to the presynaptic membrane (which is relatively a transient process) and subsequent exocytosis of the neurotransmitter, the main factor is release. Additionally, it does take a very short period of time for the neurotransmitter to diffuse across the synaptic cleft and bind to to its receptors on the post-synaptic membrane.
Firstly, we need the neurotransmitter at synaptic junctions to stimulate an action potential on the postsynaptic neurone. Continued amounts of neurotransmitter will keep Na+ diffusing in, and will keep the action potential resending when the period of refraction should be occurring.Therefore, if we do not destroy the neurotransmitter when it is not needed, a signal will keep being sent, and the effector muscle/gland will keep being stimulated.For example, touching a hot plate results may result in your bicep contracting to pull your arm away. If the neurotransmitter isn't destroyed, your bicep will keep contracting even after the event.
dopaime
Your mother
Radiant energy
To initiate a chemical reaction an activation energy is necessary.
Activation energy is the amount of energy needed to start a reaction.
solar energyLight energy is needed to initiate. This energy is stored as chemical energy
A source of Fuel Compression and an Ignition source
Yes, calcium is necessary for the release of neurotransmitters that cause a muscle to initiate a contraction. Calcium is also needed to bind to the troponin-tropomyosin complex causing it to change position so the myosin head can attach to the actin molecule which results in contraction.
I believe varicosity in the context of your question refers to the enlarged and tortuous vein, artery, or lymphatic vessel in the area of the synapse. Therefore in the aplysia synaptic junctions between the sensory and motor neurons experience long term facilition in response to 5-HT and long term depression in response to RMRFamide. Research has shown macromolecules synthesis is needed for expression of the long lasting structural changes in the sensory cells and that this synthesis is porportional with long term functional modulation of sensorimotor synapses.