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The classic presentation of neurogenic shock is hypotension caused by profound vasodilation from loss of sympathetic tone and bradycardia. The bradaycardia is typical of spinal transection above T-4. As T-4 is responsible for cardiac innervation.

Spinal shock is broken down into 4 phases

Phase one - complete loss of reflex response, sensitivity, and flaccid paralysis at and below the level of the spinal trauma.

Phase two - (day 1-3) slight return of some reflexes (i.e. stretch reflex) below site of injury

Phase three - (1 - 3weeks) Hyperreflexia occurs. this is a result of increasing numbers of post-synapitic cholinergic receptors (up regulations) and lack of feedback from the CNS below the level of the injury. (do not give depolarizing paralytics to these patients ... anesthesia/nursing pearl)

phase four -1m to 12months - continued increasing hyperreflexia w/ spacticity. nearing one year patients with spinal injuries are also at risk for autonomic hyperreflexia with spinal injuries that occur from T-6 to T-10.

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Q: What are the signs and symptoms of spinal and neurogenic shock?
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