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What else can I help you with?
Distance Gamma Hydrea is from earth?
The distance is 41 parsecs (approx 134 lightyears).
How do you get to the hydrea on Poptropica?
You recieve a starfish from the Greek museum (it will be on Poseidon's statue's head). Then you go to the god's temples and Poseidon's will be above, to the right, of Hades's. Go inside and place the starfish on the offering table, then, when the door opens, swim out into the water and dive. Get the air bubbles for oxygen, and then you will swim down the the hydra (there will be a sign). Nearby the entrance is a giant clam with a pearl, so make sure you get the pearl too. Hope this helps!
What things help you if you get a viral infection?
There are many drugs used to treat viral infections. The drugs used to treat topical viral infections such as Herpes Simplex type I are: acyclovir (zovirax), Docosanol (Abreva), penciclovir (Denavir). Drugs used to treat Herpes Zoster include Capsaicin (Capsin, Zostix, Zostix-Hp). Drugs used to treat systemic viral infections come in many categories as listed below: Drugs for Aids: -CD4 lymphocytes Nucleoside and nucleotide Inhibitor drugs for HIV/AIDS: -Abacavir (Ziagen); Adefovir (Dipivoxil); Didanosine (DDI, Videx); Emtricitabine (FTC, Coviracil); Lamivudine (3TC, Epivir); Stavudine (d4T, Zerit); Tenofovir (Viread); Zalcitabine (ddC, Hivid); Zidovudine (AZT, Aztec, Retrovir) Nonnucleoside Reverse Transcriptase Inhibitor: -Delavirdine (Rescriptor); Efavirenz (Sustiva); Nevirapine (Viramune) Protease Inhibitor: -Amprenavir (Agenerase); Indinavir (Crixivan); Neflinavir (Viracept); Ritonavir (Novir); Saquinavir (Fortovase, Invirase) Combination Drugs: -Kaletra (Lopinavir, Ritonavir); Combivir (Lamivudine/ Zidovudine); Scriptene (didanosine, Zidovudine); Trizivir (Abacavir, Lamivudine, Zidovudine) Other Drugs used for HIV/AIDS: -aldesleukin (Porleukin); Ampligen; Atevirdine mesylate; AZDU; Calanolide A; carbovir; CD4 human immunoglobulin; cytolin; Dextran Sulfate; Diethyldthiocarbamate (Imuthiol); Foscarnet (Foscavir); Gamimune N Immunoglobulin (Hivig); Hydroxyurea (Hydrea, Mylocel); Immune globulin (Gamimune N); Immupath; Interferon alfa-n3 (alferon LDO); Interferon beta-1a (Avonex, Rebif); Interferon beta-1b (Betaseron); Interleukin-10 (Tenovil); Lidakol; Multikine; Probucol (Panavir); Recombinant human CD4 (Receptin); Soluble T4; Thymopentin (Timunox); Trichosanthin; tumor necrosis factor; VaxSyn HIV - 1; Zintevir.
Chronic myelogenous leukemia (CML)?
DefinitionChronic myelogenous leukemia is cancer that starts inside bone marrow, the soft tissue inside bones that helps form blood cells. The cancer grows from cells that produce white blood cells.See also:Acute myeloid leukemia (AML)Chronic lymphocytic leukemia (CLL)LeukemiaAlternative NamesCML; Chronic granulocytic leukemia; Leukemia - chronic granulocytic (CML)Causes, incidence, and risk factorsCML most often occurs in middle-aged adults and in children. The disease affects 1 to 2 people per 100,000 and makes up 7 - 20% cases of leukemia.It is usually associated with a chromosome abnormality called the Philadelphia chromosome.Radiation increases the risk of developing CML. Exposure may occur from:High-dose radiation treatments used in the past to treat thyroid cancer or Hodgkin's lymphomaNuclear disasterIt takes many years to develop leukemia from this cause. However, most people treated for cancer with radiation do not go on to develop leukemia, and most patients with CML have not been exposed to radiation.SymptomsCML causes rapid growth of the immature blood-forming cells (myeloid precursors) in the bone marrow, blood, and body tissues.Chronic myelogenous leukemia is grouped into several phases:ChronicAcceleratedBlast crisisThe chronic phase can last for months or years. The disease may have few or no symptoms during this time. Most people are diagnosed during this stage, when they are having blood tests done for other reasons.The accelerated phase is a more dangerous phase, during which the leukemia cells grow more quickly. This phase may be associated with fever (without infection), bone pain, and a swollen spleen.If untreated, CML progresses to the blast crisis phase. Bleeding and infection may occur due to bone marrow failure. Other possible symptoms include:Bleeding and bruisingExcessive sweating (night sweats)FatigueLow-grade feverPressure under the lower left ribs from a swollen spleenSudden appearance of small pinpoint red marks on the skin (petechiae)WeaknessSigns and testsA physical examination often reveals a swollen spleen. A complete blood count (CBC) shows an increased number of white blood cells.Other tests that may be done include:Bone marrow aspiration and biopsyCBC with white blood cell differentialBlood and bone marrow testing for the presence of the Philadelphia chromosomeThis disease may also alter the results of the following tests:Platelet countUric acidTreatmentImatinib (Gleevec) is the first-line therapy for everyone with CML. Gleevec is a pill, taken by mouth. It is associated with very high rates of remission and survival. New medications similiar to Gleevec include dasatinib (Sprycel) and nilotinib (Tasigna).Sometimes a chemotherapy medicine called hydroxyurea (Hydrea) is used temporarily to reduce the white blood cell count if it is very high at diagnosis.The blast crisis phase is very difficult to treat, because it is marked by a very high count of immature white blood cells (leukemia cells). It is treated similarly to acute myeloid leukemia (AML) or acute lymphoid leukemia (ALL).The only known cure for CML is a bone marrow transplant or stem cell transplant. You should discuss your options in detail with your oncologist.Support GroupsSee:Cancer support groupLeukemia support groupExpectations (prognosis)Since the introduction of Gleevec, the outlook for patients with CML has improved dramatically. When the signs and symptoms of CML go away, you are said to be in remission. Many patients can remain in remission for many years while on this drug.Stem cell tansplantation should be considered in patients whose disease comes back after initial treatment with imatinib (Gleevec). Long-term cure after transplantation ranges from 60 - 80%.ComplicationsBlast crisis can lead to complications, including infection, bleeding, fatigue, unexplained fever, and kidney problems. Chemotherapy can have serious side effects, depending on the drugs used.Calling your health care providerCall your health care provider if you have symptoms of CML or have been diagnosed with CML and develop a fever higher than 100F, chills, sore throat, or cough.PreventionAvoid exposure to radiation when possible.ReferencesAbeloff MD, Armitage JO, Niederhuber JE, Kastan MB, McKenna WG, eds. Abeloff's Clinical Oncology. 4th ed. Philadelphia, Pa: Elsevier Churchill Livingstone; 2008:chap 107.
List of all drugs which cross blood brain barrier?
Drug ClassGeneric Name(Trade Name)AbsorptionDistributionElimination Alkylating Agentsmelphalon(Alkeran)oral - variably and incompletely absorbed from the GI tract, decreased in presence of foodmoderately high protein binding, Vz 0.5 l/kgdeactivated in plasma by hydrolysiscyclophosphamide(Cytoxan)oral - high bioavailability; intravenouscrosses blood-brain barrier (limited)hepatic biotransformation (includes activation); 5 - 25% eliminated unchanged (renal); parent and metabolites eliminated in urine are toxic to bladderifosfamide(Ifex)intravenous infusion onlyactive metabolites cross blood-brain barrier (limited)hepatic biotransformation (includes activation); 10 - 60% eliminated unchanged (increases with increasing dose - renal); parent and metabolites eliminated in urine are toxic to bladderbusolfan(Myleran)oral - completely absorbed from GI tractrapid hepatic biotransformationprocarbazine(Matulane)oral - Rapidly and completely absorbed from the gastrointestinal tract.Crosses blood-brain barrier.Hepatic biotransformation, very short elimination half-life. 70% renal elimination as metabolites.dacarbazine(Otic-Dome)Intravenous onlylimited access to CNS; low protein bindingExtensive hepatic biotransformation; 50% renal elimination (1/2 unchanged).Antibioticsdaunorubicin(Cerubidine)intravenous onlybody water, excluded by blood-brain barrierhepatic metabolism produces both active and inactive metabolites.daunorubicin or doxorubicin liposomes(Daunoxome or Doxil)intravenous infusion onlylimited to vascular fluid, animal studies indicate delivery to CNS; tissues selectively "acquire" liposomes.greatly reduced hepatic metabolism compared to un-encapsulated drugdoxorubicin(Adriamycin)intravenous onlyhigh protein binding; extensive uptake into many tissues, does not cross blood-brain barrierhepatic metabolism produces both active and inactive metabolites; tissue metabolism results in production of free radicals.idarubicin(Idamycin)intravenous infusion onlyextensive tissue binding of both native drug and metabolite; very high plasma protein bindinghepatic and extrahepatic metabolism to equipotent metabolite; elimination primarily biliary as active metabolite.plicamycin(Mithracin)intravenous infusion onlycrosses blood-brain barrier, concentrated in Kupffer cells, renal tubular cells and bone surfaceselimination is renalmitomycin (Mutamycin)intravenous onlydoes not cross blood brain barrierhepatic biotransformation, 10% eliminated in urine unchanged (% increases as dose increases)pentostatin(Nipent)intravenous onlycrosses blood-brain barrier (CSF concentrations ~10% of plasma concentrations within 24 hours). Low plasma protein bindinghepatic biotransformation, 30% - 70% eliminated in urine as unchanged drugmitoxantrone(Novantrone)intravenous infusion onlyrapid extensive distribution to tissues; high protein bindinghepatic; long half-life (due to tissue binding & slow metabolism); small fraction eliminated unchanged.dactinomycin(Cosmegen)intravenous onlydoes not cross blood brain barrierMinimal biotransformation; Elimination primarily biliary/fecal 50% unchanged (24 hours), another 10% unchanged in urine (24 hour); remainder of the drug is recovered within 1 week.Antimetabolitesfluorouracil(Adrucil)intravenous onlygood tissue penetration, crosses blood brain barrierhepatic metabolism produces 2 active metabolites and catabolism; respiratory elimination as carbon dioxide; 7 - 20% unchanged in urinecapecitabine(Xeloda)oral (pro drug)as for fluorouracilhepatic activation by conversion to 5 fluorouracil; elimination pattern as for 5 flurouracilfludarabine(Fludara)intravenous onlydistributed to whole body waterRAPIDLY dephosphylated in serum to 2-fluoro-Ara-A, then phosphorylated intracellularly to active compound. Elimination is renal, approximately 20% unchanged 2-fluoro-Ara-Amercaptopurine(Purinethol)oral - variably and incompletely absorbed from the GI tract (up to 50%)Crosses blood-brain barrier but poorly; low protein bindingHepatic metabolism for both activation and catabolism; degraded by xanthine oxidase; 7 - 40% eliminated unchanged.gemcitabine(Gemzar)intravenous infusion onlydistribution of active metabolite is limited by saturable process. Giving gemcitabine at an excess rate WASTES drug (eliminated intact before conversion).Intracellular metabolism (saturable) to active metabolites. Hepatic deamination to inactive uracil metabolite.Hormonal Oncologicstopotecan(Hycamtin)intravenous infusion onlygood tissue penetration, volumes approximately 2x body water, crosses blood brain barrierreversible pH-dependent hydrolysis to inactive moeity (low pH favors active compound), hepatic metabolism insignificant; 30% eliminated unchanged in urineleuprolide(Lupron)IM injection - 90% bioavailability; 1 month, 3 month and 4 month release formulationsdistributed to extracellular fluid volume; moderate (50%) protein binding.Metabolized to several inactive peptides. Less than 5% recovered as parent or pentapeptide metabolite.tamoxifen(Nolvadex)oral administration, bioavailability??Hepatic biotransformation with enterohepatic circulation. Prolonged elimination; Elimination primarily biliary/fecal, mostly as metabolitesMitosis inhibitorsetopside(VP16)oral - variable dose-dependent oral bioavailability (F decreases as dose increases); intravenousLow and variable into CSF, concentration differentials between normal and cancerous tissues. Very high protein binding (97%). Protein displacement interactions and hypoalbuminemia are concerns.Hepatic biotransformation; up to 50 - 60% renal elimination (2/3 as unchanged drug); remainder fecal.Othershydroxyurea(Hydrea)Well absorbed following oral administration.Crosses the blood-brain barrier (very small molecular weight).Hepatic metabolism (inactivation), 80% renal elimination within 12 hours (50% unchanged); balance eliminated from lungs as CO2paclitaxel(Taxol)intravenous onlyextensive extravascular distribution and/or tissue binding. Very high plasma protein binding.Hepatic p450 metabolism. Elimination primarily biliary / fecal. Variable renal elimination of unchanged drug.docetaxel(Taxotere)intravenous onlywidely distributed in tissues; slightly larger than body water; poor CNS penetration.Hepatic p450 metabolism. Elimination primarily biliary / fecal.cisplatin(Platinol)intravenous onlydoes not penetrate CNSrapid non-enzymatic conversion to inactive metabolites. Elimination usually expressed as recovered platinum (only 50% after 5 days), platinum detected in tissues for months.aspariginase(Elspar)intravenousintramuscularslow sequestration by reticuloendothelial system; poor CNS penetrationunknown pathway, only trace amounts appear in the urine following IV administration.Anti-toxicityamifostine(Ethyol)intravenous infusion onlywide rapid distributionmetabolised by alkaline phosphatase to active free thiol metabolite (binds cisplatin metabolites and alkylating agents and scavanges free radicals. Reaction favored in normal tissues (higher AP)dexrazoxane(Zinecard)intravenous onlydistributed to whole body water, low protein bindingseveral hepatic metabolites, intracellular metabolite may be responsible for action though this is speculative at this timemesna(Mesnex)intravenous onlyvolume of distribution approximates body water.rapid hepatic biotransformation to mesna disulfide; mesna disulfide is reduced to mesna by renal tubular epithelium, mesna binds and detoxifies metabolites of oxazophosphorines.I think it maybe useful for You :)
Sickle cell anemia?
DefinitionSickle cell anemia is a disease passed down through families in which red blood cells form an abnormal crescent shape. (Red blood cells are normally shaped like a disc.)Alternative NamesAnemia - sickle cell; Hemoglobin SS disease (Hb SS); Sickle cell diseaseCauses, incidence, and risk factorsHemoglobin is a protein inside red blood cells that carries oxygen. Sickle cell anemia is caused by an abnormal type of hemoglobin called hemoglobin S. Hemoglobin S distorts the shape of red blood cells, especially when exposed to low oxygen levels.The distorted red blood cells are shaped like crescents or sickles. These fragile, sickle-shaped cells deliver less oxygen to the body's tissues. They can also clog more easily in small blood vessels, and break into pieces that disrupt healthy blood flow.Sickle cell anemia is inherited from both parents. Sickle cell disease is much more common in people of African and Mediterranean descent. It is also seen in people from South and Central America, the Caribbean, and the Middle East.Someone who inherits the hemoglobin S gene from one parent and normal hemoglobin (A) from the other parent will have sickle cell trait. People with sickle cell trait do not have the symptoms of true sickle cell anemia.SymptomsSymptoms usually don't occur until after age 4 months.Almost all patients with sickle cell anemia have painful episodes (crises), which can last from hours to days. These crises can affect the bones of the back, the long bones, and the chest.Some patients have one episode every few years. Others have many episodes per year. The crises can be severe enough to require a hospital stay.Common symptoms include:Attacks of abdominal painBone painBreathlessnessDelayed growthand pubertyFatigueFeverPalenessRapid heart rateUlcers on the lower legs (in adolescents and adults)Yellowing of the eyes and skin (jaundice)Other symptoms include:Chest painExcessive thirstFrequent urinationPainful and prolonged erection (priapism - occurs in 10 - 40% of men with the disease)Poor eyesight/blindnessStrokesSkin ulcersSigns and testsTests commonly performed to diagnose and monitor patients with sickle cell anemia include:Complete blood count (CBC)Hemoglobin electrophoresisSickle cell testOther tests may include:BilirubinBlood oxygenCT scan or MRIPeripheral smearSerum creatinineSerum hemoglobinSerum potassiumUrinary castsor blood in the urineWhite blood cell countTreatmentPatients with sickle cell disease need ongoing treatment, even when they are not having a painful crisis. They should take supplements of folic acid (essential for producing red blood cells) because red blood cells are turned over so quickly.The purpose of treatment is to manage and control symptoms, and to limit the frequency of crises.During a sickle cell crisis, you may need certain treatments. Painful episodes are treated with pain medicines and by drinking plenty of fluids. It is important to treat the pain. Non-narcotic medications may be effective, but some patients will need large doses of narcotics.Hydroxyurea (Hydrea) is a drug some patients use to reduce the number of pain episodes (including chest pain and difficulty breathing). It does not work for everyone.Antibiotics and vaccines are given to prevent bacterial infections, which are common in children with sickle cell disease.Blood transfusions are used to treat a sickle cell crisis. They may also be used on a regular basis to help prevent strokes.Other treatments for complications may include:Dialysis or kidney transplant for kidney diseaseDrug rehabilitation and counseling for psychological complicationsGallbladder removal (if you have gallstone disease)Hip replacementfor avascular necrosis of the hipIrrigation or surgery for persistent, painful erections (priapism)Surgery for eye problemsWound care, zinc oxide, or surgery for leg ulcersBone marrow or stem cell transplants can cure sickle cell anemia. However, transplants have many risks, including infection, rejection, and graft-vs-host disease. Therefore, they are currently not an option for most patients. Also, sickle cell anemia patients are often unable to find well-matched donors.Support GroupsSickle cell anemia can cause great stress to the patient and family members. Joining a support group where members share common experiences and problems can relieve this stress.See: Sickle cell anemia - support groupExpectations (prognosis)In the past, sickle cell patients often died from organ failure between ages 20 and 40. Thanks to a better understanding and management of the disease, today, patients can live into their 50s or beyond.Causes of death include organ failure and infection. Some people with the disease experience minor, brief, infrequent episodes. Others experience severe, long-term, frequent episodes with many complications.ComplicationsAcute chest syndromeAnemiaBlindness/vision impairmentBrain and nervous system (neurologic) symptoms and strokeDeathDisease of many body systems (kidney, liver, lung)Drug (narcotic) abuseErectile dysfunction (as a result of priapism)GallstonesHemolytic crisisInfection, including pneumonia, gallbladder inflammation (cholecystitis), bone infection (osteomyelitis), and urinary tract infectionJoint destructionLeg sores (ulcers)Loss of function in the spleenParvovirus B19infection, leading to low red blood cell production (aplastic crisis)Splenic sequestration syndromeTissue death in the kidneyCalling your health care providerCall your health care provider if you have:Painful crisesAny symptoms of infection (fever, body aches, headache, fatigue)PreventionSickle cell anemia can only occur when two people who carry sickle cell trait have a child together. Genetic counseling is recommended for all carriers of sickle cell trait. About 1 in 12 African Americans has sickle cell trait. It is possible to diagnose sickle cell anemia during pregnancy.You can prevent sickling of red blood cells by:Getting enough fluidsGetting enough oxygenQuickly treating infectionsHave physical exams every 3 - 6 months to ensure that you are getting enough nutrition and activity, and that you are receiving the proper vaccinations. Regular eye exams are also recommended.PREVENTING INFECTIONSPeople with sickle cell anemia need to keep their immunizations up to date, including Haemophilus influenza, pneumococcal, meningococcal, hepatitis B, and influenza.Some patients may receive antibiotics to prevent infections.PREVENTING CRISESParents should encourage children with sickle cell anemia to lead normal lives.To reduce sickle cell crises, take the following precautions:To prevent oxygen loss, avoid: Demanding physical activity (especially if the spleen is enlarged)Emotional stressEnvironments with low oxygen (high altitudes, nonpressurized airplane flights)SmokingKnown sources of infectionTo make sure you're getting enough fluids: Avoid too much exposure to the sunHave fluids on hand, both at home and awayRecognize signs of dehydrationTo avoid infection: Consider having the child wear a Medic Alert braceletHave the child vaccinated as recommended by the health care providerShare the above information with teachers and other caretakers, when necessaryBe aware of the effects that chronic, life-threatening illnesses can have on siblings, marriages, parents, and the child.ReferencesLee MT, Piomelli S, Granger S, et al. Stroke prevention trial in sickle cell anemia (STOP): extended follow-up and final results. Blood. 2006;108:847-852.Brawley OW, Cornelius LJ, Edwards LR, Gamble VN, Green BL, Inturrisi C, et al. National Institutes of Health consensus development conference statement: hydroxyurea treatment for sickle cell disease. Ann Intern Med. 2008;148:932-938.Geller AK, O'Connor MK. The sickle cell crisis: a dilemma in pain relief. Mayo Clin Proc. 2008;83:320-323.Hebbel RP. Pathobiology of sickle cell disease. In: Hoffman R, Benz EJ, Shattil SS, et al., eds. Hematology: Basic Principles and Practice. 5th ed. Philadelphia, Pa: Elsevier Churchill Livingstone;2008:chap 42.Saunthararajah Y, Vichinsky EP, Embury SH. Sickle cell disease. Clinical features and management. In: Hoffman R, Benz Jr. EJ, Shattil SS, eds. Hematology: Basic Principles and Practice. 5th ed. Philadelphia, Pa: Churchill Livingston; 2008:chap 43.U.S. Preventive Services Task Force. Screening for Sickle Cell Disease in Newborns: U.S. Preventive Services Task Force Recommendation Statement. Agency for Healthcare Research and Quality, Rockville, MD. Sep 2007:AHRQ Publication No. 07-05104-EF-2.
How many generic prescription drugs exist?
acarbose tablet Precose®acebutolol capsule Sectral®acetazolamide tablet Diamox®acetazolamide SR capsule Diamox Sequels®acetic acid otic Acetasol®acetic acid/hydrocortisone otic Acetasol®-HCacetohexamide tablet Dymelor®acetylcysteine solution Mucomyst®acyclovir tablet, capsule, suspension Zovirax®albuterol inhalation solution, tablet, syrupProventil®alendronate sodium tablet Fosamax®allopurinol tablet Zyloprim®alprazolam tablet Xanax®amantadine capsule, syrup Symmetrel®amcinonide cream, ointment, lotion Cyclocort®amiloride tablet Midamor®amiloride/HCTZ tablet Moduretic®aminocaproic acid tablet Amicar®aminophylline tablet, liquidamiodarone tablet Cordarone®**amitriptyline tablet Elavil®amlodipine besylate/benazepril Lotrel®amlodipine besylate tablet Norvasc®amoxapine tablet Asendin®amoxicillin capsule, tablet, suspension, chews, drops Amoxil®amoxicillin/pot. clavulanate tabs, chews, suspension Augmentin®amphetamine/ dextroamphetamine tablet Adderall®anagrelide capsule Agrylin®atenolol tablet Tenormin®atenolol/chlorthalidone tablet Tenoretic®atropine ophthalmic solution Atropair®azathioprine tablet Imuran®azithromycin tablet, packet, suspension Zithromax®« Back to top »Bbacitracin ophthalmic solutionbacitracin/polymyxin ointment Polysporin®baclofen tablet Lioresal®benazepril tablet Lotensin®benazepril HCL tablet Lotensin®benzocaine/antipyrine otic Aurodex®benzonatate capsule Tessalon®benztropine tablet Cogentin®betamethasone dipropionate cream, gel, lotion, ointment Diprolene®betamethasone valerate liquid, lotion Beta-val®betamethasone/clotrimazole cream Lotrisone®betamethasone dipropionate augmented cream Diprolene® AFbetaxolol ophthalmic solutionbetaxolol tablet Kerlone®bethanechol tablet Urecholine®brimonidine ophthalmic solution Alphagan®bromocriptine tablet Parlodel®budeprion SR tablet Wellbutrin SR®budeprion XL tablet Wellbutrin XL®bumetanide tablet Bumex®bupropion SR tablet Wellbutrin SR®bupropion XL tablet Wellbutrin XL®buspirone tablet BuSpar®« Back to top »Ccabergoline tablet Dostinex®calcipotriene solution Dovonex®calcitonin-salmon nasal spray Fortical®, Miacalcin®calcitriol capsule Rocaltrol®captopril tablet Capoten®captopril/HCTZ tablet Capozide®carbamazepine tablet, chews, suspension Tegretol®carbidopa/levodopa tablet Sinemet®carisoprodol tablet Soma®carisoprodol/ASA tablet Soma® Compoundcarteolol ophthalmic solutioncarvedilol tablet Coreg®cefaclor capsule, suspension Ceclor®cefadroxil capsule, tablet, suspension Duricef®cefdinir capsule, suspension Omnicef®cefprozil tablet, suspension Cefzil®cefuroxime tablet Ceftin®cephalexin capsule Keflex®cephradine capsule Velosef®chloral hydrate capsulechlordiazepoxide capsule Librium®chlorhexidine gluconate oral rinse Peridex®chloroquine tablet Aralen®chlorothiazide tablet Diuril®chlorpromazine tablet, drops, syrup Thorazine®chlorpropamide tablet Diabinese®chlorthalidone tablet Hygroton®cholestyramine/sucrose powder Questran®choline mag trisalicylate tablet Trilisate®ciclopirox cream, gel, topical suspension Loprox®ciclopirox topical solution Penlac®cilostazol tablet Pletal®cimetidine tablet, liquid Tagamet®ciprofloxacin tablet, suspension Cipro®citalopram tablet Celexa®**citric acid/sodium citrate solution Bicitra®clarithromycin tablet Biaxin®clemastine tablet Tavist®clindamycin capsule Cleocin®clindamycin topical gel, lotion, solution Cleocin®clobetasol propionate cream, lotion, ointment Temovate®clomiphene citrate tablet* Clomid®*clomipramine capsule Anafranil®clonazepam tablet Klonopin®clonidine tablet Catapres®clorazepate tablet Tranxene®clozapine tablet Clozaril®codeine/APAP tablet Tylenol® #3colchicine tabletcolestipol HCL granules, tablet Colestid®cortisone acetate tablet Cortone®cromolyn ophthalmic solution Crolom®cromolyn neb sodium solution Intal®cryselle™ tabletcyanocobalamin injectioncyclobenzaprine tablet Flexeril®cyclophosphamide tablet++ Cytoxan®++cyclosporine capsule, solution Sandimmune®**cyclosporine, modified capsule, solution Neoral®« Back to top »Ddantrolene capsule Dantrium ®danazol capsule Danocrine®desipramine tablet Norpramin®desmopressin tablet, nasal spray, nasal solution DDAVP®**desonide/petrolatum cream Desonide®desoximetasone gel, ointment, cream Topicort®dexamethasone sod phos ophthalmic suspensiondexamethasone tablet, elixir Decadron®dextroamphetamine tablet Dexedrine®dextroamphetamine SR capsule Dexedrine Spansule®diazepam tablet, solution Valium®diclofenac sod ophthalmic solutiondiclofenac tablet Voltaren®dicloxacillin capsule Dynapen®dicyclomine capsule, tablet Bentyl®didanosine capsule Videx EC®diflorasone ointment, cream Psorcon®diflunisal tablet Dolobid®digoxin tablet, elixir Lanoxin®**diltiazem SR capsule Cardizem® CDdiltiazem SR capsule Cardizem® SRdiltiazem SR capsule Dilacor® XRdiltiazem tablet Cardizem®diphenoxylate/atropine tablet Lomotil®dipivefrin ophthalmic solution Propine®dipyridamole tablet Persantine®disopyramide & ER capsule Norpace®, Norpace CR®**dorzolamide HCL ophthalmic solution Trusopt®dorzolamide-timolol ophthalmic solution Cosopt®doxazosin tablet Cardura®doxepin capsule Sinequan®doxycycline capsule, tablet Vibramycin®dyphylline tablet Lufyllin®« Back to top »Eenalapril tablet Vasotec®enalapril/HCTZ tablet Vaseretic®ergotamine/caffeine tablet, suppository Cafergot®erythromycin base EC tablet Emycin®erythromycin base ophthalmic ointmenterythromycin base/benzoyl peroxide gel Benzamycin®erythromycin ethylsuc tablet, suspension EES®erythromycin/sulfisoxazole suspension Pediazole®estazolam tablet Prosom®estradiol tablet Estrace®estradiol transdermal patchestropipate tablet Ogen®ethambutol tablet Myambutol®ethosuximide syrup Zarontin®**etidronate disodium Didronel®etodolac capsule Lodine®etoposide capsule++ Toposar®++, Vepesid®++« Back to top »Ffamciclovir tablet Famvir®famotidine tablet Pepcid®felodipine tablet Plendil®fenofibrate capsule, tabletfenoprofen tablet, capsule Nalfon®fexofenadine Allegra®fentanyl patch Duragesic®finasteride tablet Proscar®flecainide 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