Scott, L. A., and M. S. Stone. "Viral Exanthems." Dermatology Online Journal 9 (August 2003): 4.
Beers, Mark H., MD, and Robert Berkow, MD, editors. "Viral Infections: Measles." Section 19, Chapter 265. In The Merck Manual of Diagnosis and Therapy. Whitehouse Station, NJ: Merck Research Laboratories, 2004.
There are a number of viral infections for which vaccines are available. Most viral infections, though, have no vaccine available.
Both the viral disease and the building material are spelled "shingles."
Viral vectors are modified viruses that can carry genetic material into cells. They work by infecting cells and inserting the desired genetic material into the cell's DNA. This allows the cell to produce the desired protein or carry out a specific function.
The neck of a virus, also known as the "nucleocapsid", is the structure that contains the viral genetic material (DNA or RNA). It plays a crucial role in protecting and delivering the viral genome to the host cell during infection. The neck helps ensure that the viral genetic material is efficiently released and replicated inside the host cell.
After a virus enters a host cell and releases its genetic material, the viral capsid is broken down or degraded by the cell's enzymes. The capsid proteins are typically recycled or used by the cell for its own processes. This allows the viral genetic material to be released and begin replicating inside the host cell.
Viruses inject their genetic material (DNA or RNA) into the host cell's nucleus in order to replicate. Once inside, the viral genetic material hijacks the cell's machinery to produce more viral particles.
During the process of viral replication, the virus uses host cells to produce copies of its viral DNA. This process typically involves the virus injecting its genetic material into the host cell's nucleus, where it hijacks the cell's machinery to replicate its DNA. This results in the production of multiple copies of the viral DNA, which can then be packaged into new viral particles.
Various antiviral drugs can inhibit viral replication by targeting different stages of the viral life cycle, such as attachment and entry, replication of viral genetic material, protein synthesis, and release of new virions. Additionally, the body's immune response, including interferons and antibodies, can also inhibit viral replication by neutralizing viruses and promoting their clearance.
Viruses make copies of themselves by hijacking host cells and using the cell's machinery to replicate their genetic material. The virus enters the host cell, releases its genetic material, and tricks the cell into making viral proteins and new viral particles. These new viral particles then go on to infect other cells and continue the cycle of replication.
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This process is called viral entry and occurs when the viral envelope fuses with the host cell's plasma membrane, allowing the viral genetic material and other components to enter the cell. Once inside, the virus hijacks the host cell's machinery to replicate and produce more viruses.