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between the inner mitochondrial membranes. if you mean which substrates inhibit or"bind" the 3A4 enzyme, well, my favorites are lanzoprazole, fluconazole, ritonavir. The idea is to use something else, like the substances mentioned, that have a greater affinity for 3A4, and then they use up the enzyme, making it take longer to process other substrates. Like when you want an HIV drug to stay in system longer, so you use another substrate to slow down the processing of the drug you are trying to 'boost'. Plus you still have to figure UGTs into the equation. And it also matters what and why you are 'binding". capice, or am I misinterpreting your Q?

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