Antianxiety
No.
if your referring to pregabalin (Lyrica) then the answer is no. It has no narcotic activity and in no way resembles an opiate structurally. It resembles GABA, an inhibitory neurotransmitter in the CNS.
No not yet at least. GABA as of now is illegal in some places, but that doesn't mean it's a drug.
Yes. Valium possesses anxiolytic, anticonvulsant, hypnotic, sedative, skeletal muscle relaxant, and amnestic properties. The pharmacological action of Valium enhances the effect of the neurotransmitter GABA by binding to the benzodiazepine site on the GABAA receptor leading to central nervous system depression.
An example of a drug that blocks release of a neurotransmitter is botulinum toxin (Ach) and atropine.
PCP primarily acts as an antagonist of NMDA, which is a glutamate receptor. It also affects dopamine, norepinephrine, serotonin, acetylcholine, and GABA.
There are seven major neurotransmitters that appear to account for the effects produced by the actions of drugs and alcohol abuse. These neurotransmitters include dopamine, serotonin, norepinephrine, endorphins, anandamide, glutamate, and GABA.
Opium.
Depressant Drugs: Alcohol, Benzodiazepines, Barbiturates and other central nervous system depressant drugs act primarily on a neurotransmitter substance known as GABA (Gamma Aminobutyric Acid). GABA is an inhibitory neurotransmitter that makes other neurons less likely to activate. The depressant drugs are GABA agonists, acting to help GABA reduce neuronal activation more efficiently than it usually would. Alcohol also inhibits (acts as an antagonist against) another excitatory neurotransmitter (Glutamate), making it harder for Glutamate to get the nervous system excited. Stimulant Drugs Amphetamines have their primary effects on the neurotransmitter Dopamine. Amphetamines both induce the terminal button of Dopamine-producing neurons to let more Dopamine out than normal, and also keep that Dopamine out in the synapse longer than it normally would be allowed to stay. Amphetamine also acts agonistically on receptors for a different neurotransmitter, Norepinephrine, by competing with Norepinephrine for post-synaptic receptors and turning those post-synaptic receptors on. Cocaine has its major effect by blocking the re-uptake of the neurotransmitters Dopamine and Serotonin. Opioid Drugs: Opioid drugs bind to special endorphin receptors in the brain (the 'mu', 'kappa', 'sigma' 'delta' and 'gamma' receptors) that have to do with pain. When these receptors are occupied and activated, the perception of pain lessens. Drug treatments for opioid addictions sometimes include the administration of Naltrexone, which is an opioid antagonist. Naltrexone competes with the opioids for their receptor sites, but is not itself capable of activating those receptor sites. An opioid addict on Naltrexone is thus rendered more or less incapable of getting high from their opioid drug of choice; they may take an opioid, but it will be blocked from the opioid receptors by the Naltrexone, and will not have its effect. Cannabinoids: Marijuana has a complex set of effects. It acts on the neurotransmitters Serotonin, Dopamine and Acetylcholine. It also binds to a receptor for a recently discovered neurotransmitter known as Anadamide. Hallucinogens: LSD is known to antagonize Serotonin by blocking its release.
stimulant it's a neurotransmitter, not a drug
Nicotine is one of the most common drugs that acts on the nervous system. It binds to the same receptors that acetylcholine binds to, creating a similar excitatory effect. This is why nicotine is a stimulant. Nicotine also facilitates the release of dopamine in the brain, giving smokers that relaxed, more alert feeling. Another drug that affects the nervous system is alcohol. In low doses, it acts as a stimulate, partially because it acts on glutamate receptors (glutamate is the most common excitatory neurotransmitter in the brain). However, in high doses, alcohol is a depressant, acting on GABA receptors (GABA is a major inhibitory neurotransmitter). Alcohol causes motor, cognitive, verbal, and perceptual impairments. However, nicotine is one of the most addictive substances in the United States, and both drugs have serious side effects on other areas of the body.
No. Barbiturates are derived from barbituric acid (although some "synthetic barbiturates" exist). Gabapentin is not. Barbiturates act directly on GABA-A receptors (as do in benzodiazepines--but using a different mechanism) which causes their anxiolytic and sedative properties. Gabapentin a synthetic drug, intended to mimic indigenous GABA; with the hopes (by its manufacturer) that it would act much like barbiturates and benzodiazepines. However, the actual effects of gabapentin are NOT directly on GABA-A or GABA-B receptors. How they effect the brain (and thus determining how to categorize such a drug))--well, it is simply unknown at this point by pharmacologists. Evidence is pointing to gabapentin's action as a calcium channel blocker. But in short, we don't really know. If one had to categorize it, it would be a GABA analogue (pregabalin is another drug related to gabapentin). it is distinct from the Barbiturate structure; and it acts in a different area of the brain, so it in no way shape or form would be under the "barbiturate" category.