Beta 2 adrenergic agonists cause increased potassium entry into cells, which can lead to hypokalemia
Beta 2 adrenergic agonists cause increased potassium entry into cells, which can lead to hypokalemia
Beta Agonist, corticosteroids, anticholinergic agents, mast cell stabilizers
Hypokalemia and hyperkalmia both can have effects on the heart function. Hypokalemia and hyperkalemia can cause cardiac arriythmias.
Hypokalamia.
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Patricia Marian McCaffrey has written: 'Studies of agonist and partial agonist activity at the beta-adrenoceptor in man'
It could. If its a diabetic patient who has raised serum postassium due to diabetic nephropathy then ace inhibitor can improve his diabetic nephropathy leading to hypokalemia.... BUT it DOESNT cause hypokalemia directly... instead it leads to hyperkalemia...
Hypokalemia or low potassium can cause lethal heart rhythms. general tiredness , muscle twitching and damage.
Morphine
Hypokalemia can probably cause an atrioventricular block.
Dobutamine is a potent beta-agonist with major activity on the beta-1 (heart) and weaker activity on beta-2 (blood vessels and lungs) receptors. It also has weak alpha-1 activity (blood vessels).The beta actions would cause an increase in heart rate and vasodilation. The alpha action could cause vasoconstriction but it would only be seen if the beta effects were over-ridden or opposed (for instance if the person was also on a beta blocking drug).So, technically yes dobutamine could cause vasconstriction, but clinically it is unlikely.
Usually, hyperkalemia is associated with usage of digoxin. This is due to the blocking action of digoxin on the Na/K ase which results in accumulation of extracellular K+.Most of the times, patients presenting with heart problems are already on diuretics before they are prescribed with digoxin. Diuretics cause hypokalemia as they result in excessive excretion of K+ from the body. Hypokalemia in turn causes digoxin toxicity. Digoxin toxicity does not cause hypokalemia, but hypokalemia can worsen digoxin toxicity.