decreased ATP production
ischemia is a result of hypoxia. hypoxia means low oxygen in the heart.
Anterior Wall Ischemia: [blockage in the Left Anterior Descending Artery]
Cellular ischemia occurs when perfusion (oxygenation of the body and tissues) decreases. Because of the lack of blood flow through the capillaries the metabolism goes from aerobic (with oxygen) to anaerobic (without oxygen) which causes metabolic acidosis. The blood stagnates in the capillaries and lactic acid builds up due to the pre-capillary sphincter relaxing. Also the vasomotor center fails and the carbon dioxide level increases. Because the post-capillary sphincters stay constricted the capillaries are enlarged with fluid. On and on the end result is a drop in cardiac output.
Cardiac ischemia occurs as a result of insufficient blood flow to the coronary blood vessels. This often occurs due the formation of plagues in the coronary arteries. These plagues narrow the interior of these blood flows resulting in less blood flow to the heart. Angina (or chest pain) sometimes occur as a symptom of cardiac ischemia. The heart has the highest oxygen consumption per tissue mass when compared to other organs in the human body. Blood will flow to the heart during its diastole phase. The heart's vasomotor tone is often controlled by its endothelium where healthy endothelial cells will release nitric oxide to cause vasodilation to improve blood flow. However, endothelial cells which are damaged by plague cannot produce nitric oxide as efficiently so the presence of plagues will decrease vasodilation and this will hinder blood flow. Angina will occur when the oxygen supply does not meet the heart's demand. Propanolol can be used to treat cardiac ischemia. It is a non-selective beta-blocker. It can limit the heart rate by limiting the release to catecolamines. The diastolic coronary perfusion time will hence increase and this is favorable for cardiac ischemia. It also decreases due excitability of the heart and hence reduces the myrocardial oxygen demand.
Coronary artery disease, in and of itself, does not cause pain. What causes pain is the restriction of blood flow to the cardiac muscle itself, which causes a buildup of metabolic byproducts, including lactic acid and carbon dioxide (amongst others), which results in pain from ischemia and acidosis. The acidosis and ischemia result in abnormal function in the affected muscle, which, depending on where it is, may result in other symptoms, such as shortness of breath, congestive heart failure, and rapid or irregular heart rates. Coronary artery disease is the process that most frequently leads to the blockage of blood flow, but without the restriction of blood flow, does not cause pain.
ischemia is a result of hypoxia. hypoxia means low oxygen in the heart.
Anterior Wall Ischemia: [blockage in the Left Anterior Descending Artery]
No. It results in mitochondrial adaptations
A decrease in input costs to firms in a market will result in
Cellular ischemia occurs when perfusion (oxygenation of the body and tissues) decreases. Because of the lack of blood flow through the capillaries the metabolism goes from aerobic (with oxygen) to anaerobic (without oxygen) which causes metabolic acidosis. The blood stagnates in the capillaries and lactic acid builds up due to the pre-capillary sphincter relaxing. Also the vasomotor center fails and the carbon dioxide level increases. Because the post-capillary sphincters stay constricted the capillaries are enlarged with fluid. On and on the end result is a drop in cardiac output.
A normal result is a patient that demonstrates adequate oxygenation through pulse oximetry, blood gas tests, and clinical observation.
increase in demand and decrease in supply.
Keep it dry to avoid oxygenation bond that result rust
The result is 20.04
reversible ischemia
Yes.
A general decrease in wages. - Apex