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Is a 16.6 white blood count high?
Yes. A WBC ct. of 16.6 is indicative of an infection or an acute stress response, most commonly.
Does Rheumatoid arthiritis cause WBC to be 26?
No on the contrary Normal white blood cell count is about 4.3 to 8.10. White blood cells can go down from many conditions and certain medications. The main purpose of WBCs is to fight infection so if they are decreased your body may not be able to fight off infections. Some cause include: 1. Viral infections 2. Congenital disorders 3. Cancer 4. Autoimmune disorders such as Rheumatoid arthritis. 5. Overwhelming infections that use up white blood cells faster than they can be produced 6. Drugs that destroy white blood cells The underlying cause for low WBC count needs to be found in order to determine treatment.
What happens to your intestines if you have a heart attack?
intestinal ischemia and infarction is damage to (ischemia) or death of (infarction) part of the intestine due to a decrease in its blood supply.CausesThere are several possible causes of intestinal ischemia and infarction. Hernia: If the intestine moves into the wrong place or becomes tangled, this can lead to intestinal ischemia.Adhesions: The intestine may become trapped in scar tissue from past surgery (adhesions). This can lead to ischemia if left untreated.Embolus: A blood clot from the heart or main blood vessels may travel through the bloodstream and block one of the arteries supplying the intestine. People who have had a heart attack or who have arrhythmias, such as atrial fibrillation, are at risk for this problem.Arterial thrombosis: The arteries that supply blood to the intestine may become so narrowed fromatherosclerotic disease (cholesterol buildup) that they become blocked. When this happens in the arteries to the heart, it causes a heart attack. When it happens in the arteries to the intestine, it causes intestinal ischemia.Venous thrombosis: The veins carrying blood away from the intestines may become blocked by blood clots. This blocks blood flow into the intestines. This is more common in people with liver disease, cancer, or blood clotting disorders.Low blood pressure: Very low blood pressure in patients who already have narrowing of the intestinal arteries may also cause intestinal ischemia. This typically occurs in patients who are very ill for other reasons. It can be compared to losing water pressure in a hose with a partial blockage.SymptomsThe hallmark symptom of intestinal ischemia is abdominal pain. Other symptoms include: DiarrheaFeverVomitingExams and TestsLaboratory tests may show a high white blood cell (WBC) count (a marker of infection) and increased acid in the bloodstream. There may be bleeding in the GI tract. Other tests include:AngiogramCT scan of the abdomenDoppler ultrasound of the abdomenNone of these tests are foolproof, however. Sometimes the only sure way to diagnose intestinal ischemia is with a surgical procedure.TreatmentTreatment usually requires surgery. The section of intestine that has died is removed, and the healthy remaining ends of bowel reconnected. In some cases, a colostomy or ileostomy is needed. The blockage of arteries to the intestine is corrected, if possible.Outlook (Prognosis)Intestinal ischemia is a serious condition that can result in death if not treated promptly. The outlook depends on the cause. A good outcome may be achieved with prompt treatment. Possible ComplicationsIntestinal infarction may require a colostomy or ileostomy, which may be short-term or permanent. Peritonitis is common in these cases. Some people may develop severe illness, with fever and a bloodstream infection (sepsis).When to Contact a Medical ProfessionalCall your health care provider if you have any severe abdominal pain. PreventionPreventive measures include: Control risk factors, such as irregular heartbeat, high blood pressure, and high cholesterolDo not smokeEat a nutritious dietQuickly treat herniasAlternative NamesIntestinal necrosis; Ischemic bowel; Dead bowel; Dead gut
Why is acetaminophen not very effective in treating arthritis?
First off, acetaminophen is great for arthritis. This question stem is wrong, and I'll explain why. Mediators of pain and inflammation are created in your body by an enzyme named cyclooxygenase (COX). This enzyme is therefore a target of drugs such as aspirin, ibuprofen, and perhaps acetaminophen amongst others. Aspirin and ibuprofen directly interact with this enzyme, binding it and thereby inhibiting it. Acetaminophen's mechanism is more or less unknown, but is thought to inhibit COX through another mechanism: scavenging COX's substrates. COX needs free radicals to function. These radicals are a reactant in the chemical reaction it catalyzes. Without radicals, COX cannot proceed. When our immune system is fighting an a bug, our white blood cells release radicals to kill the infection. Sometimes our white blood cells perceive ourself as foreign, and mount a similar attack. Inflammation increases free radicals. If we take acetaminophen during an inflammatory process, the drug will be overwhelmed by all of the radicals (because it binds them, remember?) If the acetaminophen is exhausted, this means COX function will be uninterrupted. This makes acetaminophen less effective in managing the pain and inflammation secondary to an inflammatory process. This being said, osteoarthritis (from degenerative changes/loss of cartilage) is NOT an inflammatory process (we don't know why steroid injections work), and so acetaminophen IS a great treatment for it. This is especially true because osteoarthritis is a long term problem that requires long term treatment, and ibuprofen or aspirin or naproxen etc. would likely cause peptic ulcers if taken that long, and so acetaminophen is a first line choice. This question stem is misleading and incorrect. Celecoxib/celebrex is an NSAID selective for cox 2 enzyme, easier on the stomach and another option for long term pain control of osteoarthritis. Acetaminophen would NOT be a great choice for arthritis from an inflammatory etiology, such as rheumatoid, or connective tissue diseases like lupus, for the reasons before explained. Acetaminophen is also not the best choice for any pain caused by tissue injury (back sprain, torn ligament etc) because of reasons explained before (excess free radical release from WBC's cleaning up the mess). There is more to acetaminophen's mechanism than we understand. It apparently doesn't inhibit COX all that much, because COX enzyme is critical in making prostaglandins that dilate the kidney's arterioles. NSAIDS are a big no no in someone who is old with already low kidney function or in someone with bad kidney disease like diabetes. However, acetaminophen is great for pain management in people with kidney malefaction because, although we believe it inhibits COX via scavenging radicals, it apparently doesn't have a big enough effect on COX to inhibit prostaglandins and cause a decrease in GFR.
What is the life span of antibodies and antigens?
Since the antibodies are a part of WBC's they ought to have a life equal to the WBC's
Which WBC take part in diapedesis?
Neutrophils, monocytes and lymphocytes
What part of the blood contains the other 3 parts?
Plasma, contains RBC, WBC and proteins
what does wbc of 20.3 indicate?
is wbc of 20.3 high
What part of the human body prouduces rbc and wbc?
The red marrow of bones makes RBCs and WBCs.
What happens when wbc increases?
wht happens if WBC increases?
What is the WBC boxing title abbreviated for?
wbc is the 'world boxing council' :)
Most numerous wbc's ina normal wbc differential?
neutrophis :)
What is the ratio of WBC to RBC?
1 to 2 WBC TO 1000 RBC
Abnormal WBC Esterase in urinalysis?
wbc esterace 2+ Abnormal occult blood 1+ Abnormal wbc 11-30 Abnormal rbc 4-10 Abnormal
How many belts does sugar ray Leonard have?
wbc welterweight title wba jr middleweight title wbc middleweight title wbc super middleweight title wbc light heavyweight title.
What is the wbc function?
The function of WBC (white blood cells) is to FIGHT INFECTIONS
