0
What else can I help you with?
What is the ion needed to initiate the release of acetylcholine into the synaptic cleft called?
The ion needed to initiate the release of acetylcholine into the synaptic cleft is calcium (Ca2+). When an action potential reaches the presynaptic terminal, it causes voltage-gated calcium channels to open, allowing calcium to enter and trigger the release of acetylcholine-containing vesicles.
What most directly causes synaptic vesicles to release acetylcholine into the synaptic cleft?
The entry of calcium ions into the presynaptic terminal triggers the fusion of synaptic vesicles containing acetylcholine with the cell membrane, leading to the release of acetylcholine into the synaptic cleft. This process is known as calcium-dependent exocytosis and is a key mechanism for neurotransmitter release at synapses.
Synaptic vesicles in the axon terminal of a motor neuron contain what neurotransmitter?
Synaptic vesicles store neurotransmitters to be released into the synapses. In the case of most motoneurons, this neurotransmitter is acetylcholine (ACh). The neurons that interface with the sympathetic nervous system, also technically motoneurons, release norepinephrine.
What is the neurotransmitter of the motor neuron?
Alpha-Motor neurons release the neurotransmitter acetylcholine at a synapse called the neuromuscular junction. When the acetylcholine binds to acetylcholine receptors on the muscle fiber, an action potential is propagated along the muscle fiber in both directions.
Preganglionic sympathetic fibers trigger the release of?
Preganglionic sympathetic fibers trigger the release of acetylcholine at the synapse with postganglionic neurons in the sympathetic ganglia. This neurotransmitter binds to nicotinic acetylcholine receptors on the postganglionic neuron, which then propagate the signal to release norepinephrine at the target tissue.
How does the botulinum toxin act in humans?
Botulinum toxin latches onto specific proteins in nerve endings and irreversibly destroys them. These proteins control the release of acetylcholine, a neurotransmitter that stimulates muscle cells.
What terminates a muscle contraction?
"A muscle cell remains contracted until the release of acetylcholine stops and an enzyme produced at the axon terminal destroys any remaining acetylcholine."
Is acetylcholine the substance released by axonal endings of the somatic efferent fibers and by the parasympathetic nerve fiber endings?
Yes, it does. In contrast, the sympathetic nerves release adrenaline
What is the chemical transmitter released at the neuromuscular junction?
Acetylcholine is the primary chemical transmitter released at the neuromuscular junction. It binds to acetylcholine receptors on the muscle cell membrane, leading to muscle contraction.
What do nerves have to do with exocytosis?
Nerves nerve endings that release neurotransmitters (eg. Acetylcholine) that initiate action potential to the innervated organ causing dpolarization, etc. This can be released through exocytosis.
What is the function of the acetylcholine sacs?
a small organic molecule liberated at nerve endings as a neurotransmitter. It is particularly important in the stimulation of muscle tissue. The transmission of an impulse to the end of the nerve causes it to release neurotransmitter molecules onto the surface of the next cell, stimulating it. After such release, the acetylcholine is quickly broken into acetate and choline, which pass back to the first cell to be recycled into acetylcholine again. The poison curareacts by blocking the transmission of acetylcholine. Some nerve gases operate by preventing the breakdown of acetylcholine causing continual stimulation of the receptor cells, which leads to intense spasms of the muscles, including the heart. Acetylcholine is often abbreviated as Ach.
Why is it important for the release of acetylcholine to stop and for enzymes to destroy any remaining acetylcholine in the neuromuscular junction?
it has caca
How does botulinum toxin help spasticity?
Botulinum toxin injections can help with spasticity by blocking the release of acetylcholine at the neuromuscular junction, reducing muscle activity and spasm. This can help improve range of motion and muscle function in individuals with spasticity.
What is the stimulus for Acetylcholine release?
The stimulus for acetylcholine release is the action potential traveling down the axon of the presynaptic neuron. This depolarization causes calcium channels to open, allowing calcium ions to enter the axon terminal and trigger the release of acetylcholine into the synaptic cleft.
Why is it so important for the release of acetylcholine to stop and for enzymes to destroy any remaining acetylcholine in the neuromuscular junction?
it has caca
Cholinergic synapses release what neurotransmitter?
acetylcholine (ACh)
What is the motor neuron ending?
Motor neuron endings are specialized structures at the terminal ends of motor neurons that form synapses with muscle fibers. These endings release neurotransmitters, such as acetylcholine, to stimulate muscle contractions. They play a crucial role in transmitting signals from the nervous system to muscles, allowing for voluntary movement.
