Alveolar ventilation may be decreased by the following factors
1.decrease in pulmonary ventlation
2.high altitude
3.blockage of the respiratory system
PaCO2.
The alveolar sac, also called an alveolus, is lined by cells called type I pneumocytes.
One physical factor that can influence respiration is temperature. Another physical factor that can influence is the diffusion constant of each gas.
type II alveolar cells
Negative pressure causes oxygen to enter the alveoli. Air is pulled into the lungs as the lungs expand. The alveoli are membranes of small balloon-like structures attached to the branches of the bronchial passages.
Decreased pulmonary and alveolar ventilation.
nothing
PaCO2.
ecause of air trapping and ineffective alveolar ventilation
function of alveolar macrophagesThe function of alveolar macrophage in the lungs is to remove dust particles and other debris from alveolar spaces. -SheshiAveolar macrophages work to break up surfactanct in the aveoli which exists to decrease surface tension in the lungs. If there is a disorder where the macrophage does not function correctly, such as in PAP, the surfactant will not be broken up and decreased ventilation will occur resulting in less oxygen delivery.
alveolar ventilation
alveolar ventilation.
AVR=(Tidal Volume-Dead Space)* Respiratory Rate(or Frequency)
Alveolar volume is lung capacity. Alveoli are the air sacs in the bronchioles. How much air the lungs hold during both inspiration and expiration is lung capacity which is alveolar volume. Hope this helps
Rapid increase of organic acids in the body you would expect to observe the increase in your heart rate. You would also observe increased alveolar ventilation, decreased blood pH and increased in blood pressure.
The main function of the pulmonary vein is to carry oxygenated blood from the lungs to the left atrium. Then it is transferred to the aorta, which takes the blood to the head and arms.
•The normal airway is distended by alveolar attachments during expiration, allowing alveolar emptying and lung deflation. In COPD, these attachments are disrupted because of emphysema, thus contributing to airway closure during expiration, trapping gas in the alveoli and resulting in hyperinflation