93923 Multi site and lead
93922 Single site and lead
Rebreathing can lead to a higher PCO2 because it involves inhaling the already exhaled air, which contains higher levels of carbon dioxide. This reduces the exchange of fresh oxygen from the environment, causing an accumulation of carbon dioxide in the respiratory system and increased PCO2 levels.
blood entering the lungs has a partial pressure of oxygen (PO2) of 40 mmHg and a partial pressure of carbon dioxide (PCO2) of 46 mmHg; alveoli, on the other hand, have a PO2 of 105 mmHg and a PCO2 of 40 mmHg. As the blood moves past the alveoli, oxygen and carbon dioxide will diffuse down their respective partial pressure gradients. Oxygen will move from the alveolar space (PO2 of 105 mmHg) to the blood stream (PO2 of 40 mmHg). Carbon dioxide will move from the blood (PCO2 of 46 mmHg) to the alveolar space (PCO2 of 40 mmHg). As the blood leaves the alveolus, the PO2 and PCO2 will have essentially equilibrated with the alveolar air.
No, it is higher or the CO2 would not move out of the lungs.
Increases due to greater oxygen demands and a rising blood CO2 concentration (PCO2).
-Decreased pH -Increased Temperature -Increased Pco2
This is called the Bohr effect where a increase in pC02 which decrease the pH leads to a decreased affinity of hemoglobin to oxygen. This means that hemoglobin unloads oxygen in areas where pC02 is high e.g. active tissue and that the binding coefficient of hemoglobin is highest in the lung where pC02 is negligible.
pco2
In pulmonary arteries, PO2 is around 40 mmHg and PCO2 is around 46 mmHg. In pulmonary veins, PO2 is around 100 mmHg and PCO2 is around 40 mmHg. In systemic arteries, PO2 is around 100 mmHg and PCO2 is around 40 mmHg. In systemic veins, PO2 is around 40 mmHg and PCO2 is around 46 mmHg.
The units for pCO2 are typically expressed in millimeters of mercury (mmHg) or in kilopascals (kPa).
constrict
Teflon is used for the membrane of pco2 electrodes as it allows for the diffusion of co2 but not ions.
PCO2