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The effect of the toxin is to block the release of inhibitory neurotransmitters (glycine and gamma-amino butyric acid (GABA) across the synaptic cleft, which is required to check the nervous impulse. The consequence of this is dangerous overactivity in the muscles is the failure of inhibition of motor reflexes. Normally your nervous system keeps this in check.If nervous impulses cannot be checked by normal inhibitory mechanisms, it produces the generalized muscular spasms characteristic of tetanus.

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Q: How does tetanospasmin affect motor control?
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