0
What else can I help you with?
What is the difference between an acute and non acute fracture?
A break in a bone from a quick, one-time injury
What kind of angle are there?
Right angle (90) Acute angle (<90) Obtuse angle (>90) Straight angle (180) Reflex angle (>180)
How do you use over-acuteness in a sentence?
I ask for more acute for math
Is holmium poisonous?
Yes holmium has a low level of acute toxicity.
A result of 60 in an ESR test what it means?
it shows acute or chronic infection.
What are the two major causes of ATN?
ATN Acute Tubular Necrosis The causes of acute renal failure (ARF) are conventionally and conveniently divided into 3 categories: prerenal, renal, and postrenal. Prerenal ARF involves an essentially normal kidney that is responding to hypoperfusion by decreasing the glomerular filtration rate (GFR). Renal, or intrinsic, ARF refers to a condition in which the pathology lies within the kidney itself. Postrenal failure is caused by an obstruction of the urinary tract. Acute tubular necrosis (ATN) is the most common cause of ARF in the renal category.
What is necrosis of burned skin?
Acute, non healing burn
What does a high or low specific gravity of urine signify?
Increases in specific gravity (hypersthenuria, i.e. increased concentration of solutes in the urine) may be associated with dehydration, diarrhea, emesis, excessive sweating, urinary tract/bladder infection, glucosuria, renal artery stenosis, hepatorenal syndrome, decreased blood flow to the kidney (as a result of heart failure), and excess of antidiuretic hormone caused by Syndrome of inappropriate antidiuretic hormone. A specific gravity greater than 1.035 is consistent with frank dehydration. Decreased specific gravity (hyposthenuria, i.e. decreased concentration of solutes in urine) may be associated with renal failure, pyelonephritis, diabetes insipidus, acute tubular necrosis, interstitial nephritis, and excessive fluid intake (e.g., psychogenic polydipsia).
What is your elomalacia in a Dog?
Myelomalacia is the softening or necrosis of the spinal cord after an acute injury.
Can you take expired levaquin?
As long as you have a kidney in line For a transplant if you need it. It would not be a good idea for your health. Expired tetracycline has been known to cause acute tubular necrosis (basically your kidneys die). So I guess for short, no.
What is the difference between coagulative and liquefactive necrosis?
Liquefactive necrosis is good for your body, especially your brain (due to the beneficial liquids produced) but coagulative necrosis is bad due to the clumping and coagulation (i.e. clotting) that occurs.Coagulation necrosis is the "acute" necrosis in which the protein fibers become denatured and everything turns into a semi-solid mess of dead tissue. Liquefactive necrosis is a more "chronic" necrosis in which the dead tissue is digested into a liquid which can then be removed by the macrophages.
What is fatty necrosis?
Fat necrosis is one of many types of necrosis. Necrosis is cell death with inflammation (different from apoptosis, which is without inflammation). Fat necrosis occurs in two forms. 1. Traumatic fat necrosis is the result of trauma. Cellular damage to fat-rich organs, such as the breasts, can lead to necrosis of the adipocytes. 2. Fat necrosis as a result of acute haemorrhagic pancreatitis. In this case, damage to the pancrease releases lipolytic enzymes into the blood, causing damage, and eventual necrosis of adipocytes.
What is the damage to the nephron caused by lack of blood flow called?
The damage caused to the nephron due to lack of blood flow is called ischemic injury or acute tubular necrosis. This condition occurs when there is inadequate oxygen and nutrients reaching the nephrons, leading to their damage and dysfunction. Early recognition and treatment are important to prevent further kidney damage.
What are the names of the acute angles?
Acute angles do not have specific names.
How do you spell gangeours gallbladder?
The infection with gangrene (from tissue necrosis or specific bacteria) is a complication of acute cholecystitis, and may be called a gangrenous gallbladder or gangrenous gall bladder. Because rupture of the bladder would release toxins into the entire abdomen, this is a critical medical situation.
Acute tubular necrosis?
DefinitionAcute tubular necrosis is a kidney disorder involving damage to the tubule cells of the kidneys, resulting in acute kidney failure.Alternative NamesNecrosis - renal tubular; ATN; Necrosis - acute tubularCauses, incidence, and risk factorsAcute tubular necrosis (ATN) is caused by lack of oxygen to the kidney tissues (ischemia of the kidneys).The internal structures of the kidney, particularly the tissues of the kidney tubule, become damaged or destroyed. ATN is one of the most common structural changes that can lead to acute renal failure.ATN is one of the most common causes of kidney failure in hospitalized patients. Risks for acute tubular necrosis include:Blood transfusion reactionInjury or trauma that damages the musclesRecent major surgerySeptic shock or other forms of shockSevere low blood pressure (hypotension) that lasts longer than 30 minutesLiver disease and kidney damage caused by diabetes (diabetic nephropathy) may make a person more susceptible to the condition.ATN can be caused by:Exposure to medications that are toxic to the kidneys (such as aminoglycoside antibiotics)Antifungal agents (such as amphotericin)Dye used for x-ray (radiographic) studiesSymptomsDecreased consciousnessComaDelirium or confusionDrowsy, lethargic, hard to arouseDecreased urine output or no urine outputGeneral swelling, fluid retentionNausea, vomitingNote: Other symptoms of acute kidney failure may also be present.Signs and testsExamination usually indicates acute kidney failure. There may be signs of fluid overload, including abnormal sounds on listening to the heart and lungs with a stethoscope (auscultation).Other signs include:BUN and serum creatinine levels may increaseFractional excretion of sodium and of urea may be relatively highKidney biopsymay show acute tubular necrosis (but a biopsy is rarely done)Urinalysis may show casts, kidney tubular cells, and red blood cellsUrine sodiummay be highUrine specific gravity and osmolarity urine indicate dilute urineTreatmentIn most people, acute tubular necrosis is reversible. The goal of treatment is to prevent life-threatening complications of acute kidney failure during the time the lesion is present.Treatment focuses on preventing the excess build-up of fluids and wastes, while allowing the kidneys to heal. Patents should be watched for deterioration of kidney function.Treatment can include:Identifying and treating the underlying cause of the problemRestricting fluid intake to a volume equal to the volume of urine producedRestricting substances normally removed by the kidneys (such as protein, sodium, potassium) to minimize their buildup in the bodyTaking medications to help control potassium levels in the bloodstreamTaking water pills (diuretics) to increase fluid removal from the kidneyDialysis can remove excess waste and fluids. This can make you feel better, and may make the kidney failure easier to control. Dialysis may not be necessary for all people, but is often lifesaving, especially if serum potassium is dangerously high.Dialysis may be needed in the following cases:Decreased mental statusFluid overloadIncreased potassium levelsPericarditisTotal lack of urine productionUncontrolled buildup of nitrogen waste productsExpectations (prognosis)The duration of symptoms varies. The decreased urine output phase may last from a few days to 6 weeks or more. This is occasionally followed by a period of high urine output, where the healed and newly functioning kidneys try to clear the body of fluid and wastes.One or two days after urine output rises, symptoms reduce and laboratory values begin to return to normal.ComplicationsChronic renal failureEnd-stage renal diseaseGastrointestinal loss of bloodHypertensionIncreased risk of infectionCalling your health care providerCall your health care provider if your urine output decreases or stops, or if you develop other symptoms of acute tubular necrosis.PreventionPromptly treating conditions that can lead to decreased blood flow and/or decreased oxygen to the kidneys can reduce the risk of acute tubular necrosis.Blood transfusions are crossmatched to reduce the risk of incompatibility reactions.Control conditions such as diabetes, liver disorders, and cardiac disorders to reduce the risk of acute tubular necrosis.Carefully monitor exposure to medications that can be toxic to the kidney. Have your blood levels of these medications checked regularly. Drink a lot of fluids after having any radiocontrast dyes to allow them to be removed from the body and reduce the risk of kidney damage.ReferencesGoldman L, Ausiello D. Cecil Medicine. 23rd ed. Philadelphia, Pa: Saunders; 2007 Chapter 121
What is uric acid nepropathy?
Acute uric acid nephropathyOverproduction of uric often occurs when tissue breakdown is accelerated. Acute uric acid nephropathy is the term used to describe the development of acute oligoanuric renal failure caused by renal tubular obstruction by urate and uric acid crystals.
