ADH does not exactly cause the vasoconstriction but increases the peripheral vascular resistancewhich results increases of arterial blood pressure
norepinephrine, epinephrine and angiotensin II
Yes
High blood pressure is the most common cause of prolonged vasoconstriction in the kidney.
I believe they are stimulants due to the fact that cocaine is a stimulant and it causes vasoconstriction.
Deficiency of adh
The sympathetic nervous system primarily causes vasoconstriction in the body.
Low levels of ADH can cause excessive urination (polyuria) followed by extreme thirst (polydipsia). A rare water metabolism disorder, called central diabetes insipidus, is sometimes the cause of ADH deficiency but low ADH doesn't not cause diabetes insipidus.
Vasoconstriction is regulated by the autonomic nervous system and various vasoactive substances including norepinephrine, angiotensin II, and endothelin. These substances act on smooth muscle cells in blood vessel walls to cause contraction, leading to vasoconstriction.
No, decreased levels of antidiuretic hormone (ADH) cause the nephrons to become less permeable to water. ADH promotes the reabsorption of water in the kidneys by increasing the permeability of the collecting ducts. When ADH levels are low, less water is reabsorbed, leading to more dilute urine and increased water excretion. Thus, decreased ADH results in reduced water retention in the body.
Antidiuretic hormone (ADH), also known as vasopressin, primarily affects the kidneys by promoting water reabsorption in the collecting ducts, which helps concentrate urine and reduce water loss. It also influences blood vessels, causing vasoconstriction, which can increase blood pressure. Additionally, ADH can impact the brain by modulating social behavior and bonding. Overall, its primary role is to maintain fluid balance and regulate blood pressure.
the high osmolarity of blood acts upon the osmoreceptor of hypothalmus cause the release of adh low plasma volume increase plasma cholecystokinin
No, sympathetic activity typically causes vasoconstriction, not vasodilation.