Yes it does.
An increase in blood sugar levels cause the release of the hormone insulin by the pancreas. Insulin then lowers this blood sugar level restoring it to original non-lethal blood glucose levels.
Cells located in the pancreas that produces insulin. Insulin controls the amount of glucose in the blood and, when glucose levels spike, cause certain cells to 'suck up' the access glucose and store it.
Insulin takes glucose from the blood and converts it glycogen that can be stored in the liver and muscles.Insulin can increase blood pressure in a reaction called metabolic syndrome. It decreases glucose and developing studies show that developing diabetes may increase calcium levels which can affect artery calcium deposits. These deposits are associated with high blood pressure.
It is lowered by insulin, which is produced in the beta cells of the pancreas in an area called the Islet of Langerhans. Blood glucose is also suppressed by somatostatin which is produced in the delta cells of the pancreas.
Increased blood sugar will cause insulin production to rise and the cells will store the excess sugar, increased blood pressure causes a slower heart rate and decrease in oxygen levels will increase red blood cell production.
The pancreas releases two hormones, insulin and glucagon. When blood sugar is high, insulin is released to promote uptake of glucose into the body's cells. Oppositely, when blood sugar is low, glucagon is released to stimulate breakdown of glycogen which is the body's store of sugar in the liver to raise blood glucose levels.
decreased Insulin level will result in increased levels of blood glucose or in other terms diabetes mellitus
When glucose levels are high in the human body, a condition known as hyperglycemia occurs. The pancreas responds by releasing insulin, a hormone that helps cells absorb glucose for energy or storage. If high glucose levels persist, it can lead to insulin resistance, where cells become less responsive to insulin, potentially resulting in diabetes. Prolonged hyperglycemia can also cause damage to blood vessels and organs.
Insulin is the chemical released from the pancreas (in non-Diabetics) to lower blood sugar whenever we eat. Type 1 Diabetics are insulin dependent (need to inject themselves with insulin to stay at a healthy blood sugar level) and Type 2 Diabetics who don't necessarily look after themselves well may need to become insulin dependent.
Insulin causes the glucose in your blood to enter the cells for energy. It does not cause the liver to change glucose into anything. Your liver does, however, store extra sugar in the form of glucagon.
Insulin is secreted when blood sugar rises. It works by stimulating receptors on tissue cells to escort glucose into the cells. It all suppresses glucagon, hormone-sensitive lipase (a hormone responsible for mobilizing and using fat stores) and suppresses the appetite centers in the brain. Glucagon is secreted when blood sugar is low. It triggers gluconeogenesis in the liver, which means that fats and proteins are converted into glucose and released into the blood. It also is responsible for mobilizing glycogen stores, suppresses insulin, and mobilizes fat stores. So glucagon and insulin not only do opposite things, they are antagonistic to each other, meaning when one is high the other one is low. They do not technically work "together". High levels of insulin are common in those who consume excessive calories and excessive carbohydrate foods. For those who are sensitive to the effects of insulin, this can cause reactive hypoglycemia and obesity.
Diabetes is a disease where the body cannot independently manage its blood glucose levels. This is mainly due to the inadequate production of a hormone called insulin. Therefore, insulin injections would be a suitable treatment for diabetics because it gives them enough insulin to maintain their blood glucose levels.