Organopesticides will increase the severity of muscle contraction beyond normal conditions. Acetylcholine esterase inhibition will allow a more than normal amount of acetylcholine to bind to receptors.
potato
It doesn't atropine only acts on muscarinic receptors (it's a competitive antagonist here for ACh), but histamine acts on different receptors (histamine receptors). There is no direct interaction between atropine and histamine receptors
the receptors on the postsynaptic membrane
Yes. Gandora's Ignition Effect is a destruction effect, so you can chain Stardust Dragon to it, to negate and destroy it.
Death and destruction
Martine J. Smit has written: 'Chemokine receptors as drug targets' -- subject(s): Drug Delivery Systems, Chemokines, Cell receptors, Chemokine Receptors, Drug effects, Effect of drugs on, Receptors
yes skeletal muscle stretch by means of contraction.
Yes, beta blockers produce a negative inotropic effect, which means they decrease the force of contraction of the heart muscle. By blocking beta-adrenergic receptors, these medications reduce the heart's responsiveness to catecholamines like adrenaline, leading to decreased myocardial contractility. This effect can be beneficial in conditions such as hypertension and heart failure, where reducing the workload on the heart is desired.
The parasympathetic division of the autonomic innervation of the heart releases acetylcholine from its varicosities (the sites where neurotransmitter is released). The acetylcholine binds to M-2 muscarinc receptors to mediate the negative chronotropic (slowing of heart rate) effect. This also mediates a negative inotropic (lowering of force of contraction) effect.
change in shape of lens.
Alot of destruction