The major symptoms involved in EDS classical type are the skin and joints
EDS classical type is inherited in an autosomal dominant manner
Excessively loose joints are the hallmark of this EDS type
Medical therapy relies on managing symptoms and trying to prevent further complications. There is no cure for EDS
Excessively loose joints are the hallmark of this EDS type
Arthrochalasia type EDS is now referred to as classic type EDS. It is a rare genetic disorder that affects the connective tissues in the body, resulting in joint hypermobility, skin hyperextensibility, and tissue fragility.
EDS (Ehlers-Danlos syndrome) is a group of genetic connective tissue disorders that affect the skin, joints, and blood vessels, leading to hypermobility and tissue fragility. ERS (Ehlers-Danlos syndrome hypermobility type) is a subtype of EDS that specifically manifests with joint hypermobility and related symptoms without significant skin or tissue involvement.
The Vascular type of Ehlers-Danlos Syndrome (EDS) used to be called EDS Type IV. It is characterized by fragile blood vessels, increased risk of organ rupture, and easy bruising. It is caused by a mutation in the COL3A1 gene, which is responsible for producing collagen type III.
Kyphoscoliosis type
Signs/symptoms in the classical form (type 1) develop in infancy and progress slowly, with death occurring in late adolescence or early adulthood. The connatal form (type 2) also develops in infancy, but progresses more rapidly
Ehlers-Danlos type 3 is the hypermobility form of the Ehlers-Danlos Syndrome. Affects 1 in 10,000 to 15,000 and is caused by an autosomal dominant or autosomal recessive mechanism. Extreme flexibility is the hallmark of this type. It is one of the most surviable types of EDS.
Chlorpromazine (Thorazine) is a classical antipsychotic agent - the first one ever used in a widespread manner, beginning in the 1950's.
A blood type is not a condition or illness. Therefore, a blood type does not and can not have "symptoms".