Severe hyperphosphatemia can cause paralysis, convulsions, and cardiac arrest
Hyperphosphatemia is generally asymptomatic
Hyperphosphatemia can lead to hypocalcemia by binding to calcium and forming insoluble calcium-phosphate complexes. This can result in symptoms such as muscle cramps, tetany, and seizures. Additionally, hyperphosphatemia can cause a decrease in serum magnesium levels due to increased renal excretion of magnesium.
Transient hyperphosphatemia is usually fairly benign
Hyperphosphatemia occurs at serum phosphate levels above 5 mg/dL
Yes
Yes
Having too much Phosphorus in the body
Yes
The disorder occurs concurrently with hypocalcemia
Yes
Too high phosphorus levels in the blood are known as hyperphosphatemia. This condition can be caused by various factors such as kidney disease, certain medications, or excessive dietary intake of phosphorus. It is important to monitor and manage phosphorus levels to prevent complications.
The recommended dosage of calcium acetate for patients with hyperphosphatemia is typically 667 mg to 2,000 mg with each meal, up to a maximum of 15,000 mg per day. It is important to follow the specific instructions provided by a healthcare provider.