Worldwide there are various treatments suggested by the medical world but none scientifically proves any solid results. The general idea amongst ophthalmologists is that there is no cure for Retinitis Pigmentosa. Proposed treatments go from a vitamin based medication to avoiding strong day light, to ozone or electro-stimulation. The truth is that some treatments do actually have certain effects. They tend to slow down the progress of the disease. But there is no real over all treatment for patients. All these treatments are a strain on the patients already hard every day life and in the end bring no solution.
Since the early 50ies the Cuban Doctor Orfilio Pelaez Molina dedicated a life time of medical research to the disease Retinitis Pigmentosa. As a young doctor he was angered when a good friend of his was diagnosed with RP and the local doctor simply said there was nothing he could do: his friend would become gradually blind.
Retinal deterioration is often referred to as retinal degeneration. This condition occurs when the cells in the retina begin to deteriorate, leading to vision loss or impairment. Age-related macular degeneration and retinitis pigmentosa are examples of retinal degenerative diseases.
The average age of onset is 28 years.
Biological Age
The age of onset of CMT can vary anywhere from young childhood to the 50s or 60s.
The usual age of onset is between 25 and 80 years of age, with a peak age of onset at 40 years old. However it can rarely occur in those in teenage years, as i myself have TOS at 15.
The age of onset ranges from 1 to 14 years with 75% starting between 7-10 years.
You can be diagnosed with schizophrenia at any age, although an age below 13 is termed "juvenile-onset schizophrenia" and an age above 45 is termed "adult-onset schizophrenia". The average age of onset for men is 18 and the average age of onset for women is 25.
The most common age at onset is the second and third decades in women and the seventh and eighth decades in men.
DefinitionRetinitis pigmentosa is an eye disease in which there is damage to the retina. The damage gets worse (progresses) over time.Alternative NamesRPCauses, incidence, and risk factorsRetinitis pigmentosa commonly runs in families. The disorder can be caused by a number of genetic defects.The cells controlling night vision (rods) are most likely to be affected. However, in some cases, retinal cone cells are damaged the most. The main sign of the disease is the presence of dark lines in the retina.As the disease gets worse, side (peripheral) vision is gradually lost. The condition may eventually lead to blindness, but usually not complete blindness. Signs and symptoms often first appear in childhood, but severe vision problems do not usually develop until early adulthood.The main risk factor is a family history of retinitis pigmentosa. It is an uncommon condition affecting about 1 in 4,000 people in the United States.SymptomsDecreased vision at night or in low lightLoss of side (peripheral) visionLoss of centralvision (in advanced cases)Signs and testsTests to evaluate the retina:Color visionExamination of the retina by ophthalmoscopy after the pupils have been dilatedFluorescein angiographyIntraocular pressureMeasurement of the electrical activity in the retina (electroretinogram)Pupil reflex responseRefraction testRetinal photographySide vision test (visual field test)Slit lamp examinationVisual acuityTreatmentThere is no effective treatment for this condition. Wearing sunglasses to protect the retina from ultraviolet light may help preserve vision.Controversial studies have suggested that treatment with antioxidants (such as vitamin A palmitate) may slow the disease from getting worse.Several clinical trials are in progress to investigate new treatments for retinitis pigmentosa.Microchip implants that go inside the retina are in the early stages of development for treating blindness associated with this condition.It can help to see a low-vision specialist. Make regular visits to an eye care specialist, who can screen for cataracts or retinal swelling -- both of which can be treated.Expectations (prognosis)The disorder will continue to progress, although slowly. Complete blindness is uncommon.ComplicationsPeripheral and central loss of vision will eventually occur.Patients with retinitis pigmentosa often develop cataracts at an early age. Cataracts can be removed if they cause vision loss.Many other conditions have similarities to retinitis pigmentosa, including:Friedreich's ataxiaLaurence-Moon syndrome (also called Laurence-Moon-Bardet-Biedl syndrome)MucopolysaccharidosisMuscular dystrophy (myotonic dystrophy)Usher syndrome (a combination of retinitis pigmentosa and hearing loss)Calling your health care providerCall your health care provider if night vision becomes difficult or if you develop other symptoms of this disorder.PreventionGenetic counseling may determine whether your children are at risk for this disease.ReferencesSieving PA. Retinitis pigmentosa and related disorders. In: Yanoff M, Duker JS, Augsburger JJ, Azar DT. Ophthalmology. 2nd ed. Philadelphia, PA: Mosby; 2004:chap 108.
Episodic paroxysmal hemicrania occurs in both sexes, with a slight female preponderance (1.3:1). The age of onset is variable; studies show EPH onset is 12-51 years.
The Classical age of ancient Greece was when the Greek culture thrived.
Golden ages are marked by great achievements. They are like a classical age, except that the classical age came first and set the standards for following golden ages. The golden age builds on the classical one. A classical age marks a high point in the nation's history