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Decreased blood flow to cardiac muscle that only injures the tissue creates a condition known as what?
Myocardial infarction is the decreased blood flow to cardiac muscle that only injures the tissue.
How does muscle fiber stretch affect myocardial contractility?
Muscle fiber stretch affects myocardial contractility through the Frank-Starling mechanism, which states that an increase in the length of cardiac muscle fibers (due to increased venous return) enhances their contractile force. When myocardial fibers are stretched, the optimal overlap between actin and myosin filaments occurs, leading to more effective cross-bridge formation during contraction. This results in stronger heart contractions and improved stroke volume. However, excessive stretching can lead to decreased contractility and heart dysfunction.
What other body systems does myocardial infarction affect?
Myocardial infarction (MI), or heart attack, primarily affects the cardiovascular system but can also impact other body systems. The respiratory system may be compromised due to reduced oxygenation and pulmonary congestion. Additionally, the nervous system can be affected, as MI may lead to stress responses and changes in brain function. Furthermore, the gastrointestinal system can experience disturbances due to decreased blood flow and stress-related issues.
What is the difference between ischaemia and infarction?
Ischemia=decreased oxygen/nutrients Infarction=no blood flow to the area ischemia can leads to infarction. ischemia means, reduced of blood supply to specific organ. while, infarction refers to death tissue.
What is the term for decreased blood supply?
Ischemia is the medical term meaning decreased blood supply. Prolonged ischemia can lead to infarction.
What happens to end diastolic volume when an increase in heart rate is not accompanied by an increase in contractility?
end diastolic volume is decreased
What happens to end diastolic volume when an increase in contractility is not accompanied by an increase in heart rate?
end diastolic volume is decreased
How does myocardial infarction lead to heart failure?
Myocardial infarction (MI) can lead to heart failure in numerous ways. First of all, early on, the heart muscle does not contract well because it is not receiving enough oxygen and other necessary substrates, so stroke volume is decreased, which may lead to congestive heart failure. Later on, if the MI is aborted with thrombolytics or with a cardiac catheterization and thrombectomy, the myocardium may be stunned, or hibernating, because of the lack of oxygen, and may return to normal function over time. If the MI completes, the portion of cardiac muscle that was affected is dead. Depending on how significant a portion that is, losing the muscle alone may result in heart failure. The dead portion of the heart will later turn into a scar, which is noncontractile and also does not allow for the normal stretch, so it may affect preload and contractility in that way as well, decreasing cardiac output and possibly leading to congestive heart failure.
How would a problem associated with the walls of the heart affect the systemic circulation?
A problem with the walls of the heart, such as cardiomyopathy or a myocardial infarction, can lead to impaired contraction and relaxation of the heart muscle. This dysfunction can reduce the heart's ability to pump blood effectively into the systemic circulation, resulting in decreased blood flow to organs and tissues. Consequently, this can cause symptoms like fatigue, shortness of breath, and fluid retention, ultimately compromising overall cardiovascular health. Additionally, reduced cardiac output can lead to compensatory mechanisms, such as increased heart rate and vascular resistance, which may further strain the heart.
What does decreased radiotracer uptake in the anterior and anteroseptal wall of heart mean?
Decreased radiotracer uptake in the anterior and anteroseptal wall of the heart typically indicates reduced blood flow or perfusion in those regions, often associated with ischemia or myocardial infarction. This finding suggests potential coronary artery disease or damage to the heart muscle in those areas. Further evaluation, such as stress testing or angiography, may be warranted to assess the underlying cause and severity of the condition.
What is subendocardium?
There don't seem to be a lot of good definitions of "subendocardium" but most references describe the subendocardial layer as that supplied by the coronary arteries deep in the myocardium. Literally, it means below the endocardium so presumably is the portion of the myocardium closest to the endocadium, which is incidentally the region most likely to be affected by myocardial infarction due to the decreased cardiac blood supply in this area.
Do beta blockers produce a negative inotropic effect?
Yes, beta blockers produce a negative inotropic effect, which means they decrease the force of contraction of the heart muscle. By blocking beta-adrenergic receptors, these medications reduce the heart's responsiveness to catecholamines like adrenaline, leading to decreased myocardial contractility. This effect can be beneficial in conditions such as hypertension and heart failure, where reducing the workload on the heart is desired.
