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Platelets are a certain type of cell present uniformly in the bloodstream. Platelets are responsible for the healing action of injuries and the provision of fibrin threads to seal the wounds. Platelets are red in color and oval in shape.
Platelets, also known as thrombocytes, are small cell fragments that play a crucial role in blood clotting. Unlike most cells in the body, platelets do not have a nucleus. This lack of a nucleus allows platelets to be more flexible and better able to change shape to form blood clots in response to injury or bleeding.
Platelets are small, disc-shaped cells that aid in blood clotting. Their shape allows them to adhere to damaged blood vessel walls and come together to form a plug at the site of injury, helping to stop bleeding. The disc shape also helps platelets flow easily through blood vessels and interact with other blood cells.
platelets.
Platelets help heal your wound, the scabs that form are the platelets
platelets.
platelets that are smaller in size than your average platelets... Can be associated with aplastic anemia.
The only formed elements that are not spherical are red blood cells (erythrocytes) which are biconcave in shape, and platelets (thrombocytes) which are irregularly shaped discs. Both of these cell types play important roles in the body's functions such as oxygen transport (red blood cells) and blood clotting (platelets).
Platelets which are pale yellow and irregular in shape and which are deposited on a wound in order to clot the blood it resembles the colour of a pus.
because there is only the defects in function of platelets in hemophilia and not reduction in number of platelets!
Here are the three processes for platelet plug formation:1. Platelet adhesionThe platelets will adhere to the collage fibres exposed in a damaged blood vessel. This occurs when the glycoprotein 1b (GPb1) receptors of the platelets interact with the von Willebrand factor (vWb) synthesised by endothelium.2. Platelet activationThis process is initiated by the collagen fibres, the adenosine diphosphate (ADP), and thrombin. It causes the platelets to synthesise thromboxane A2 (TXA2) and to releasethe contents of their dense granules which contain ADP and 5-hydroxytryptamine.3. Platelet aggregationThe released mediators will cause the platelets to change from a discoid shape to a sphere with spiny projections. This new shape will increase the chances of the platelets making contact with other platelets. The platelets will then aggregate with fibrinogen and vWf as connecting agents via their glycoprotein IIb/IIIa receptors. ADP and TXA2 act as chemotactic factors to attract more platelets and the positive feedback loop created forms the platelet plug which temporarily seals the break in the damaged blood vessel.
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