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Antiviral medications are designed to target specific proteins or enzymes unique to the virus, which are not found in host cells. This selectivity allows the medication to inhibit viral replication without harming the host cell. Additionally, antiviral medications are often metabolized or eliminated from the body before they can affect host cells.
No, Mycobacterium tuberculosis does not produce exotoxins. It mainly causes disease through its cell wall components and induces an inflammatory response in the host.
No, drug-susceptible cells and drug-resistant cells are equally likely to infect a new host.
A virus attaches to a host cell by recognizing and binding to specific proteins or receptors on the cell surface. This attachment is necessary for the virus to enter the host cell and begin the process of infection.
The specific viral proteins on the surface of the virus determine its attachment to host cell membrane receptors. These proteins bind to complementary host cell receptors, allowing the virus to attach and enter the host cell.
Viruses do not have a habitat in the traditional sense, as they are not considered living organisms. They require a host cell to replicate and survive. Once inside a host, viruses can exist in various parts of the body, depending on the type of virus and its specific target cells.
Yes, preventing the specific attachment of a virus to its host cells can be an effective way to prevent infection. By blocking the receptors on the host cell that the virus recognizes for attachment, the virus is unable to enter the cell and replicate, thus reducing the likelihood of infection. This can be achieved through various means, such as developing drugs or vaccines that target the attachment process.
Host Cell
The specific host cell for West Nile Virus is the neuronal cell in the central nervous system. Once inside these cells, the virus can replicate and cause damage, leading to neurological symptoms in infected individuals.
The protein structure of a virus typically includes specific proteins on its outer surface that help it attach to receptors on host cells. This attachment is crucial for the virus to gain entry into the host cell, infect it, and replicate. The binding specificity between viral proteins and host cell receptors is a key determinant of the virus's ability to infect specific cell types.
The virus enters the host cell and uses the cell's machinery to replicate itself.
Proteins on the surface of a virus interlock with specific receptor proteins on the host cell's plasma membrane. This interaction is crucial for the virus to gain entry into the host cell and initiate its replication cycle.