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I believe that it is beneficial to be heterozygous for the condition, since a slight mutation of the CFTR protein still occurs, provinding resistance to diseases such as typhoid fever and cholera. This is because the pathogens for the fever cannot enter through the mutated CFTR protein in the intestinal epithelium. This has increased (or rather "not decreased") the rate of carriers, and therefore the faulty recessive allele is "still around"

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14y ago

What else can I help you with?