Corticosteroids have some agonistic effects on Mineralocorticoids (aldosterone) thus dumping potassium and absorbing sodium.
Beta 2 adrenergic agonists cause increased potassium entry into cells, which can lead to hypokalemia
Hypokalemia and hyperkalmia both can have effects on the heart function. Hypokalemia and hyperkalemia can cause cardiac arriythmias.
Hypokalamia.
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It could. If its a diabetic patient who has raised serum postassium due to diabetic nephropathy then ace inhibitor can improve his diabetic nephropathy leading to hypokalemia.... BUT it DOESNT cause hypokalemia directly... instead it leads to hyperkalemia...
Hypokalemia can probably cause an atrioventricular block.
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Hypomagnesemia can result in hypokalemia and thereby cause cardiac arrhythmias
hyperkalemia as it may cause heart arrythmias and sudden death, where hypokalemia generally just causes malaise, muscle weakness and tetany. hyperkalemia as it may cause heart arrythmias and sudden death, where hypokalemia generally just causes malaise, muscle weakness and tetany.
Beta 2 agonists cause hypokalemia by stimulating the beta 2 adrenergic receptors in the skeletal muscle, liver, and kidneys, leading to increased cellular uptake of potassium. This effect can result in decreased serum potassium levels and can be exacerbated in patients who are predisposed to hypokalemia due to conditions such as diuretic use or metabolic alkalosis.
Usually, hyperkalemia is associated with usage of digoxin. This is due to the blocking action of digoxin on the Na/K ase which results in accumulation of extracellular K+.Most of the times, patients presenting with heart problems are already on diuretics before they are prescribed with digoxin. Diuretics cause hypokalemia as they result in excessive excretion of K+ from the body. Hypokalemia in turn causes digoxin toxicity. Digoxin toxicity does not cause hypokalemia, but hypokalemia can worsen digoxin toxicity.
No steroids doesnt but it makes female have beards!