0
What else can I help you with?
Increased secretion of aldosterone would result in a .... blood ph....?
Decreased blood pH. Aldosterone increases sodium reabsorption in the kidneys, leading to increased hydrogen ion secretion as a compensatory mechanism to maintain electrolyte balance. This can result in acidosis, leading to a lower blood pH.
What conditions result in renal retention of sodium?
Conditions that can result in renal retention of sodium include heart failure, liver cirrhosis, and kidney diseases such as nephrotic syndrome. In these conditions, the kidneys may become less responsive to the hormone aldosterone, which normally helps regulate sodium excretion. This leads to an accumulation of sodium in the body, which can contribute to fluid retention and high blood pressure.
How might the production of a hormone affect the expression of genes in a eukaryotic cell?
The hormone will migrate to the nucleus and bind to its counter-part-sequence some-where within the chromosomal material, thereby effecting a gene's control sequences which then, in turn, affects the expression of the Gene-in-Question.
Which of the following most likely would be an immediate result of growth factor binding to its receptor?
An immediate result of growth factor binding to its receptor would likely be the activation of intracellular signaling pathways. This could lead to various cellular responses such as changes in gene expression, cell proliferation, differentiation, or survival.
What would a rise in angiotensin II levels would result in?
A rise in angiotensin II levels can lead to vasoconstriction, increased blood pressure, and stimulation of the release of aldosterone from the adrenal glands, which increases sodium and water reabsorption in the kidneys. Ultimately, this can contribute to hypertension and increased fluid retention in the body.
What would happen if a hormone receptor did not bind to the right site?
I'm assuming you're asking what would happen if a receptor did not bind the proper hormone. The answer is a complex one because binding to a receptor does not necessarily mean that the receptor will be activated. Sometimes binding causes receptor inhibition; other times it can mean that the properties of the receptor change so that other hormones have an easier/harder time binding and activating it. But for the sake of giving an answer, let's say that we want to know what happens if a hormone binds and activates the wrong receptor. That answer is a relatively simple one: in most cases, the same events would take place that normally happen when the correct hormone binds the receptor. Let's take an example of a relatively uncommon cause of hypertension called hypertension exacerbated in pregnancy. In this condition, there's a mutation in the receptor for the hormone aldosterone that allows other hormones besides aldosterone (eg, progesterone) to bind it and activate it. When progesterone levels are high, as in pregnancy, the extra progesterone binds and heavily activates the aldosterone receptor, and the receptor essentially "thinks" that aldosterone has bound. So the action of progesterone at the aldosterone receptor are the same as aldosterone itself; since aldosterone is a major contributor to blood pressure, blood pressure increases to very high levels.
Is oral testosterone a steroid?
Testosterone is a steroid hormone from the androgen group and is found in mammals, reptiles,[1] birds,[2] and other vertebrates. In mammals, testosterone is primarily secreted in the testes of males and the ovaries of females, although small amounts are also secreted by the adrenal glands. It is the principle male sex hormone and an anabolic steroid.
Steroid hormones exert their action by?
steroid hormones can pass easily through the plasma membrane, the hormones then bind to intracellular receptors which induce a metabolic pathway which later promotes transcription of a specific gene.
What is andosterone?
Andosterone is created in the liver by metabolizing testosterone. The result is an essentially inactive male sex steroid hormone. It is one of two androgens that affect masculinity in a developing fetus.
What Hypofunctioning of the adrenal cortex leads to what condition?
Low adrenal functioning of the cortex leads to the cortex not making enough steroid hormones. It has two names: Primary AI or Addison (or Addison's) disease. In this rare condition, the adrenal glands do not work properly and cannot make enough cortisol (a "stress" hormone).
If hormones travel in the bloodstream why don't all tissues respond to all hormones?
The tissue/cells need a receptor that can interact with that hormone in order to respond to it. This receptor may be on a cell membrane, inside the cell, or even inside the nucleus (in the case of steroid hormones, for example.) Like many organic chemicals in the body, they have structures which result in certain parts of the molecule being presented to cells (active sites). Because cells and tissue have different structures as well, only certain cells will react to the presence of a particular hormone. Many hormones have antagonist hormones that cause an opposite effect. This helps control negative feedback when the target organ's hormone levels are too high.
Increased secretion of aldosterone would result in a .... blood ph....?
Decreased blood pH. Aldosterone increases sodium reabsorption in the kidneys, leading to increased hydrogen ion secretion as a compensatory mechanism to maintain electrolyte balance. This can result in acidosis, leading to a lower blood pH.
What are toxic cataracts?
Toxic cataracts result from chemical toxicity, such as steroid use.
Are the sore breasts in the earliest stages of pregnancy a result of the HCG hormone?
Yes there are. Every pregnancy symptom is a result of the HCG hormone hun.
What is to much of a growth hormone called?
Too much growth hormone would result in Gigantism.
How are the mechanisms of action of frusemide and losartan in treating hypertension different?
Frusemide is a loop diruetic. It prevents the reabsorption of NaCl at the ascending limb of the loop of Henle. Less sodium will result in less water in retained in the body so the blood volume decreases. Although the mechanisms of diruetics is not exactly known, it is believed that it causes a reduction in blood volume which leads to decreased blood pressure. It is also believed that reducing the amount of Na+ in the body leads to a reduction of intracellular Ca2+, resulting in the muscles being less receptive to endogenous vasoconstrictors. Losartan is an angiotensin receptor blocker. It blocks the angiotensin I receptor. This leads to less angiotensin II being produced. Angiotensin II itself is a vasodilator and it can also cause aldosterone and the antidiruetic hormone to be released. Both aldosterone and the antidiruetic hormone leads to the kidney to retain salt and this leads to more water being retained in the body resulting in a greater blood volume and a greater blood pressure. As Losartan inhibits angiotensin I receptors, it can prevent the release of angiotensin II and hence these effects.
Unusual weight gain or loss and sterility can result from inadequate rest or dehydration or anabolic steroid use or excessive heat loss?
C. Anabolic Steroid Use
