Yes, thiazide diuretics will decrease renal excretion of phosphate. Thiazide diuretics is used to treat high blood pressure and edema of the liver, heart, and kidney disease.
Diuretics promote urine secretion and help to decrease fluid retention. They work by increasing the excretion of water and electrolytes by the kidneys, thus leading to increased urine production. Examples of diuretics include thiazides, loop diuretics, and potassium-sparing diuretics.
Hyperphosphatemia can lead to hypocalcemia by binding to calcium and forming insoluble calcium-phosphate complexes. This can result in symptoms such as muscle cramps, tetany, and seizures. Additionally, hyperphosphatemia can cause a decrease in serum magnesium levels due to increased renal excretion of magnesium.
Usually, hyperkalemia is associated with usage of digoxin. This is due to the blocking action of digoxin on the Na/K ase which results in accumulation of extracellular K+.Most of the times, patients presenting with heart problems are already on diuretics before they are prescribed with digoxin. Diuretics cause hypokalemia as they result in excessive excretion of K+ from the body. Hypokalemia in turn causes digoxin toxicity. Digoxin toxicity does not cause hypokalemia, but hypokalemia can worsen digoxin toxicity.
urine volume excretion, as the kidneys would excrete more water to maintain balance.
Glucose levels in urine typically increase after meals when blood glucose levels rise. Glucose excretion in urine is typically higher when blood glucose levels are elevated, such as in uncontrolled diabetes.
Parathyroid hormone (PTH) is responsible for increasing the renal excretion of phosphate by acting on the kidneys to decrease phosphate reabsorption and promote its excretion in the urine.
Diuretics promote urine secretion and help to decrease fluid retention. They work by increasing the excretion of water and electrolytes by the kidneys, thus leading to increased urine production. Examples of diuretics include thiazides, loop diuretics, and potassium-sparing diuretics.
decrease edema
it decreases blood volume and preload
Because The energy is used for things like respiration and excretion. 10% of the biomass is used up from excretion.
ph will become elevated and blood will be more acidic
It controls Calcium and phosphate level.It decrease phosphate level and increase calcium level.
They are not inherently either. They can be good if they are used properly. For example, because diuretics increase urine output and decrease blood volume, if someone with high blood pressure takes some prescribed diuretics they can decrease their blood volume and thus their blood pressure. This can be very beneficial. However, if someone already has a blood pressure that is too low, possibly after they have been bleeding out, then taking diuretics would further decrease their BP and this could result in insufficient blood flow to the brain resulting in syncope, brain damage, or death
Hyperphosphatemia can lead to hypocalcemia by binding to calcium and forming insoluble calcium-phosphate complexes. This can result in symptoms such as muscle cramps, tetany, and seizures. Additionally, hyperphosphatemia can cause a decrease in serum magnesium levels due to increased renal excretion of magnesium.
Usually, hyperkalemia is associated with usage of digoxin. This is due to the blocking action of digoxin on the Na/K ase which results in accumulation of extracellular K+.Most of the times, patients presenting with heart problems are already on diuretics before they are prescribed with digoxin. Diuretics cause hypokalemia as they result in excessive excretion of K+ from the body. Hypokalemia in turn causes digoxin toxicity. Digoxin toxicity does not cause hypokalemia, but hypokalemia can worsen digoxin toxicity.
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Calcitonin decreases the calcium level.It also lowers the phosphate level.