No, atropine is not an antidote for cyanide poisoning. The primary antidotes for cyanide include hydroxocobalamin and sodium thiosulfate. Atropine is an anticholinergic medication used to treat bradycardia and certain types of poisoning, but it does not counteract the effects of cyanide.
Several antidotes are: hydroxocobalamin, sodium thiosulfate, sodium nitrite etc.
use amyl nitrate, sodium nitrate and sodium thiosulfate, each used in order to achieve the stability in the victim. the FDA-approved cynokit is an antidote that contains hydroxocobalamin, an intravenous drug. :) but double check on the computer or some thing!
Antidotes for cyanides are: sodium thiosulfate, hydroxoycobalamin, sodium nitrite, 4-dimethylamino phenol etc.
It can take minutes to hours for an individual to die from an inhaled dose of hydrogen cyanide if no antidote is given. The exact time can vary depending on factors like the concentration of the gas inhaled and the individual's overall health. Immediate medical treatment is crucial in such cases.
Cyanide poisoning occurs because cyanide inhibits the enzyme cytochrome c oxidase, which is one of the enzymes that enable cells to use oxygen. Antidotes for cyanide poisoning tend to focus on binding the cyanide ion so that this inhibition stops. The liver is capable of metabolizing cyanide as well, to take care of any small residual amounts remaining.There are several ways to accomplish this. Nitrites and/or 4-dimethylaminophenol both convert hemoglobin to methemoglobin, which binds tightly with cyanide. This causes problems of its own, though ... methemoglobin cannot carry oxygen, and it's necessary to convert a lot of hemoglobin to methemoglobin to be sure there's an excess to bind all the cyanide, which leaves the person treated with methemoglobinemia ... which then must be treated separately (usually by treatment with methylene blue). Thiosulfates react with cyanide to convert it to thiocyanide, which is much less toxic. However, the reaction does not occur quickly in the body, so this treatment must be supplemented with one of the others (usually nitrates/4-DMAP). Cyanide is present in one form of vitamin B12, so treatment with the cyanideless form hydroxocobalamin will bind cyanide in the harmless cyanocobalamin. Cobalt ions in general will bind cyanide, but cyanocobalt complexes are themselves generally toxic, so this is not necessarily much of an improvement. It is fast, though, and is sometimes used (in the form of the dicobalt EDTA complex) in cases of severe cyanide poisoning. Administration with glucose helps mitigate the toxic effects of the cyanocobalt complexes.
The cyanide antidote is nitrite, specifically amyl nitrite or sodium nitrite, which helps to convert hemoglobin to methemoglobin, allowing it to bind cyanide. 2-PAM chloride (pralidoxime) is an antidote for organophosphate poisoning. Atropine is used to treat certain types of poisoning or overdose, but not specifically for cyanide poisoning.
Atropine
Physostigmine is the antidote for atropine poisoning. It works by reversing the effects of atropine on the body's nervous system. It should be administered under medical supervision due to potential side effects.
Banerica Peroxide Duo.
The antidote for excess acetylcholine is atropine, which acts as a competitive antagonist at cholinergic receptors. Atropine inhibits the effects of acetylcholine by blocking its binding, therefore reversing toxicity symptoms such as bradycardia, bronchoconstriction, and excessive salivation caused by excess acetylcholine.
The antidote for atropine is physostigmine, a reversible inhibitor of the enzyme acetylcholinesterase. It increases the levels of acetylcholine at neurotransmitter junctions, counteracting the effects of atropine, which is an anticholinergic agent. Physostigmine is particularly effective in cases of atropine toxicity, helping to restore normal cholinergic activity in the body. However, it should be used with caution, as it can also lead to cholinergic side effects.
Physostigmine inhibit AchE (the enzyme that hadrolyse Ach), so Ach accumulate at synaptic cleft and banish the effect of atropine
Atropine and pralidoxime chloride
The antidote for physostigmine is atropine. Physostigmine is an acetylcholinesterase inhibitor that can lead to excessive acetylcholine accumulation, causing symptoms like muscle spasms and respiratory distress. Atropine, an anticholinergic drug, helps counteract these effects by blocking acetylcholine receptors. It's important to administer atropine carefully and monitor the patient's response.
atropine sulfate is used as an antidote of anticholinesterase meds administered on patients with myasthenia gravis. Atropine sulfate should be also made available when performing the Tensilon Test to reverse the effect of Edrophonium injection.
The antidote for pyridostigmine, which is a reversible acetylcholinesterase inhibitor, is atropine. Atropine works by blocking the effects of excess acetylcholine at muscarinic receptors, alleviating symptoms of cholinergic toxicity. In severe cases, pralidoxime may also be administered to help restore normal function of acetylcholinesterase if there is significant inhibition.
Several antidotes are: hydroxocobalamin, sodium thiosulfate, sodium nitrite etc.