Down syndrome results from a nondisjunction during cell division. That is, homologous chromosomes 21 fail to separate in meiosis I, or sister chromatids 21 during meiosis II or mitosis.
Down syndrome is caused by trisomy of chromosome 21 (an extra chromosome to the usual two), which is an autosomal chromosome. Therefore it is not a sex linked disorder.
Sexlinked and recessive.
Cooley's anemia, also known as beta-thalassemia, is an autosomal recessive genetic disorder. This means that both copies of the gene must be mutated in order for the disease to be present. It is not a sex-linked trait.
Thalassemia is carried on chromosome 11 and chromosome 16, depending on the type. The alpha-thalassemia gene is located on chromosome 16, while the beta-thalassemia gene is found on chromosome 11. Mutations in these genes affect the production of hemoglobin, leading to the various forms of thalassemia.
Color blindness is a recessive trait, meaning that an individual needs to inherit two copies of the gene for color blindness (one from each parent) in order to be color blind. If an individual inherits only one copy of the gene, they are considered a carrier and will not exhibit color blindness.
No, thalassemia is a genetic blood disorder that affects the production of hemoglobin in red blood cells. Blood group and Rh factor are determined by different genetic markers and are not directly linked to thalassemia.
Sexlinked and recessive.
If you have beta thalassemia trait and your partner has sickle cell trait there is a 25% chance of your child having sickle beta thalassemia.
Yes, Thalassemia sex is a linked trait. Ha ;)
Nope! The disease is equally in both males and females. This means that it is autosomal.
Thalassemia trait, also known as alpha or beta thalassemia minor, is a genetic condition characterized by a reduced production of hemoglobin. Individuals with this trait typically have normal or slightly reduced hemoglobin levels, and they do not usually suffer from iron deficiency. Therefore, taking iron supplements can lead to iron overload, potentially causing damage to organs, as the body does not require additional iron in this condition. It's essential for individuals with thalassemia trait to consult healthcare professionals before taking iron supplements.
have seen one of my friends sister had kid and all normal no complications...not thalassemia either.........beta and alpha i think there is 25% normal chance
It is unlikely.
you have a 50/50 chance me and my twin both had it and my 2 older brothers are fine 1 is a carrier and the other is not.
Thalassemia Intermedia is a recessive trait inherited disease of the red blood cells. In thalassemia, the genetic defect results in reduced rate of synthesis of normal hemoglobin chains.
Thalassemia is classified mainly into two types: alpha thalassemia and beta thalassemia, based on which globin chain is affected. Alpha thalassemia occurs due to mutations in the alpha-globin genes, while beta thalassemia results from mutations in the beta-globin genes. Each type can further be categorized into various subtypes based on the severity of the condition, such as thalassemia minor (trait) and thalassemia major (Cooley's anemia). The classification helps determine the appropriate management and treatment strategies for affected individuals.
Thalassemia trait, typically a genetic condition characterized by reduced hemoglobin production, usually doesn't worsen with age because it is a stable genetic trait. Individuals with thalassemia trait often maintain sufficient hemoglobin levels for normal function and experience few, if any, symptoms throughout their lives. While some may develop mild anemia, this does not typically progress to more severe forms or complications as they age. Regular check-ups can help manage any potential issues, but the trait itself remains stable.
No, thalassemia is not a dominant trait; it is inherited in an autosomal recessive manner. This means that a person must inherit two copies of the mutated gene, one from each parent, to exhibit the disease. Individuals with only one copy of the mutated gene are carriers and typically do not show symptoms.