Well im just going to type what it says in my book
In bacterial viruses, the cycle of the viral infection, replication, and destruction is called the lytic cycle.
During an infection, some viruses stay inside the cells but do not make new viruses. Instead of producing virus particles, the viral gene is inserted into the host chromosome and is called a provirus. Whenever the cell divides, the provirus also divides, resulting in two infected host cells. In this cycle, called the lysogenic cycle, the viral genome replicates without destorying the host cell. In some lysogenic viruses, change in the environment can causethe provirus to begin the lytic cycle, which means the destruction of the host cell.
I hope this helps ^.^ --Pengi-Chan
In the lysogenic cycle, the virus's genetic material integrates into the host's genome and remains dormant, only activating later to enter the lytic cycle. The lytic cycle involves the virus immediately taking over the host cell's machinery to replicate and destroy the host cell to release new viral particles.
Rhinovirus typically follows a lytic cycle, where it infects host cells, replicates rapidly, and causes cell lysis to release new viral particles. This cycle does not involve a latent or lysogenic phase seen in some other viruses.
Measles goes through the lytic life cycle, where the virus enters a host cell, replicates, and then destroys the host cell to release new viral particles. This leads to symptoms associated with the infection.
I believe it is lytic. Think: colds are fast acting; they don't sit in your cell for years on end. This means they are lytic (fast acting).
The Norwalk virus (Norovirus) does not have a lyosgenic cycle. It does not remain dormant as lysogenic viruses can. It is lytic and is considered virulent as many lytic viruses are. Most bacteriophages are lysogenic. See link below:
Yes rabies is lytic. The lytic cycle is a cycle of viral reproduction and is how some diseases are spread.
They both are viral replication processes :3
They both are viral replication processes :3
The lytic cycle involves the immediate replication of the virus and eventual destruction of the host cell, while the lysogenic cycle involves the integration of the virus's genetic material into the host cell's genome, leading to longer-term dormancy. Lytic cycle results in rapid production of new viral particles, while lysogenic cycle allows the virus to replicate along with the host cell's DNA until a trigger induces the lytic cycle.
both virus attaches to host cell, viral replication cycle
In the lysogenic cycle, the virus's genetic material integrates into the host's genome and remains dormant, only activating later to enter the lytic cycle. The lytic cycle involves the virus immediately taking over the host cell's machinery to replicate and destroy the host cell to release new viral particles.
HPV infects the dermis layer of cells using the lysogenic cycle.
Lysogenic !
Lysogenic.
Lytic and lysogenic cycles are primarily regulated by the genetic and environmental factors that influence viral replication. In the lytic cycle, viruses express early genes that promote replication and cell lysis, while in the lysogenic cycle, viral DNA integrates into the host genome and is regulated by repressor proteins that prevent lytic gene expression. The decision between these cycles is often influenced by environmental stressors, which can trigger the transition from lysogenic to lytic, a process known as induction. Additionally, the presence of specific host factors and the state of the host cell can also play crucial roles in determining the cycle a virus will enter.
The virus that causes AIDS, HIV, is lytic in nature. Once it attaches itself into a host cell, it will go about integrating its genetic material into the host cell and use its machinery to force the cell to make copies of the virus. Additionally, the viral cell will kill the host cell in the process.
A virus in a lysogenic cycle can suddenly enter the lytic cycle due to various stressors or environmental triggers, such as UV radiation, chemical exposure, or changes in host cell conditions. These factors can activate specific viral genes that initiate the lytic phase, leading to the production of new viral particles and eventual host cell lysis. Additionally, factors like the immune response of the host organism can also prompt the switch from lysogenic to lytic activity.